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{{Talk header}}
== Plagiarism? ==
{{WikiProject banner shell|collapsed=yes|class=B|vital=yes|1=
Interesting! I wrote/edited much of the text for Fructose in Wikipedia and can guarantee it was original. I sweated over it, trying to write it for general audience and added all the references. It is eerie to see one's work on another website, and can guarantee that mrsci.com are the plagiarists.
{{WikiProject Food and drink|importance=mid}}
[[User:Jwanderson|Jwanderson]] 06:00, 6 April 2006 (UTC)
{{WikiProject Chemicals|core|importance=High}}
{{WikiProject Molecular Biology|MCB=yes|MCB-importance=High|attention=no}}
}}


== Presence in semen, and as proof in rape cases ==


Semen is the only human product that contains any appreciable amount of fructose. For this reason it is a "marker" in rape cases. The logical place in Wikipedia for this information is in this article.[[Special:Contributions/38.104.70.30|38.104.70.30]] ([[User talk:38.104.70.30|talk]]) 22:47, 24 February 2023 (UTC)captcrisis
Sorry to but in here but this article appears wholesale at


== Talk topic on contradictory information from EFSA's perspective? ==
http://www.mrsci.com/Nutrition/Fructose.php


In Potential health effects section, there is a Self-contradictory template that states:
with a claim and reserve of copyright
looks like someone has been a naughty boy.


{{Blockquote
either this material has been plagiarised by mrsci or the author here or the author is reserving rights at mrsci and releasing rights under the GFDL here.
|{{Self-contradictory|about=the EFSA's perspective, in summary, regarding fructose, please compare to the lead|1=section|date=March 2023}}
}}


I'm guessing [[Talk:Fructose/Archive 2#Mischaracterization of the %22European Food Safety Authority%22 paper|this is the mentioned talk post]], which was [https://en.wikipedia.org/enwiki/w/index.php?title=Talk:Fructose&diff=prev&oldid=1141423162 archived back in Feb 2023], before the Mar 2023 mentioned in the template.
Tut Tut


Should a new topic be started, or the existing one unarchived, or the template removed? As a casual user, I found it a bit confusing to figure out where to get more details, with there just being the one topic above that was left unarchived. But also not familiar enough with WP editing to know if it's normal to reference archived talk posts in the article. [[User:Pb7280|Pb7280]] ([[User talk:Pb7280|talk]]) 23:10, 11 May 2023 (UTC)


:The challenge from the March 2023 talk page about whether a controversy exists within the EFSA opinion was raised by an IP editor whose interpretation was incorrect. There is no controversy.
==Some comments on the comments below:==
:I removed the "contradicton box" with other minor edits, [https://en.wikipedia.org/enwiki/w/index.php?title=Fructose&diff=prev&oldid=1154362574 here.] [[User:Zefr|Zefr]] ([[User talk:Zefr|talk]]) 00:19, 12 May 2023 (UTC)


== Relative sweetness: weight or mole basis? ==
"''What do you mean that some people react badly to fructose??"''


It is important to say whether sweetness is compared on a molar or weight basis. If the basis is weight as I suspect, the enhanced sweetness of fructose over glucose could be explained by the fact that its molecular weight is about half that of sucrose. Hydrolysis of sucrose would have a smaller effect on sweetness, as only half the weight of sucrose is converted to fructose with greater sweetness, while the other half is converted to glucose with lesser sweetness.[[User:Eaberry|Eaberry]] ([[User talk:Eaberry|talk]]) 15:47, 18 January 2024 (UTC)
The usual obvious complaint about fructose is when excess is consumed that is not absorbed in the small intestine... it leads to gas production in the bowels and water retention... bloating, flatulence and even diarrhea. See comments in Health Effects. This is well established and well known in the medical literature.


:Did you mean 'sweetness of fructose over '''sucrose?''' That would be logical. However, the following reference says that fructose is twice as sweet on a molar basis:
The health effects on people are profound and ubiquitous. Most age-related chronic diseases seem to be induced by fructose consumpion. I have studied this extensively.
:by DT Cason · 1987 · Cited by 46 — Fructose is a very sweet sugar and on a molar basis is twice as sweet as sucrose ...
:I haven't read enough to have confidence in that answer so I haven't edited the article. [[User:SciberDoc|SciberDoc]] ([[User talk:SciberDoc|talk]]) 15:08, 27 February 2024 (UTC)


== Edit of table needed, please help if you have the skill required ==
"''Why would fructose cause obesity if it's slower to digest than sacharose and also more sweeter for less calories, and also les likely to drive any addiction,''"


Table 1. Sugar content of selected common plant foods (g/100g
The article cited on obesity was only one of many. Some poorly controlled human experiments of short duration have failed to show that fructose consumption contributes to obesity. Generally the experimental failure is to not monitor total fructose consumption, which is the relevant variable; not, for example, the fructose consumption from soda pop only (a well known study did this recently, but still showed a small fructose-obesity linkage).
The headings 'Fruits', 'Vegetables' and 'Grains' need deleting. They are inappropriate and in the wrong places. As the table can usefully be sorted, any division into fruits vegetables and grains is bound to go wrong depending which way it is sorted (unless each section sorted separately which would make comparisons harder between items in different categories).


I attempted to delete the headings but everything I tried broke the table display when I tried it in the sandbox. Help from someone skilled with tables would be appreciated. Thanks.
Fructose causes obesity in two ways. First, fructose tips the leptin/ghrelin balance toward fat deposition as opposed to energy production. The upshot of this is that two people eating the same number of calories may have different results... the fructose eater will slowly become more obese than a non-fructose eater on the same caloric intake, other things being equal. This effect is little noted because nearly everyone eats significant fructose; and that is 'normal.' The hormonal balance shift caused by fructose is thought to be a genetic adaptation, since in most of human history high fructose foods would have been available primarily in the late summer through fall when one would preferentially be storing some fat for the winter.
[[User:SciberDoc|SciberDoc]] ([[User talk:SciberDoc|talk]]) 14:57, 27 February 2024 (UTC)


== Absolute sweetness of fructose is identical at 5 °C as 50 °C? ==
Second, fructose doesn't induce the satiety response; yet it does contain calories. This, combined with the induced hormonal shift, typically results in overconsumption and obesity. Today, when high fructose foods are eaten year-round in unprecedented quantities, people are getting obese and suffering metabolic syndrome (aka syndrome X)and other age-related chronic diseases as a result.


I was not able to find this information in the cited source (Lee, Thomas D. (1 January 2000). "Sweeteners". Kirk-Othmer Encyclopedia of Chemical Technology. doi:10.1002/0471238961.19230505120505.a01.pub2. ISBN 978-0471238966.). Does anyone know where did this information come from? [[Special:Contributions/182.255.32.46|182.255.32.46]] ([[User talk:182.255.32.46|talk]]) 04:04, 30 August 2024 (UTC)
This article shows a positive correlation for "corn syrup" consumption and poor association for fats or carbohydrates. This very positive association existed even though they chose "corn syrup" as an indicator of "refined carbohydrate" consumption. This reflects the popular notion that "refined carbohydrates" are damaging because of poor nutrition; and does not recognize the unique role of fructose in de-regulation of appetite and satiety responses.
[http://www.ajcn.org/cgi/reprint/79/5/774]
Since their "corn syrup" category includes both high fructose corn syrup (usually containing 55% fructose and 45% glucose) and plain corn syrup (typically 95%+ glucose), the effect of fructose is understated. Based on the animal studies I have reviewed, the correlation would be even more positive for total fructose consumption, since fructose appears to be the root cause, and it's effects are masked by the categories chosen for this study.


:Ok I found it, but in the "sugars" section, not "sweeteners". In fact I suspect that most of the citations of this source should be "sugars". The sweeteners part barely talked about fructose at all. [[Special:Contributions/182.255.32.46|182.255.32.46]] ([[User talk:182.255.32.46|talk]]) 04:10, 30 August 2024 (UTC)
"''...and also les likely to drive any addiction,...''"
I think this is just a gratuitous comment. If you have any references to support it I would be interested.

''"because it's often absorbed with fruits it's associated with many highly valuable nutriments!"''

That fructose is associated with other desirable nutrients, such as the anti-oxidants in/near the peel of fruits, doesn't make it a healthful molecule to eat. Even in fresh fruit, eaten whole, the antioxidants only partially offset the oxidants, such as hydrogen peroxide, produced as side-products from glycations passing through the various reactions as they become Advanced Glycation End Products (AGEs. Fructose is especially unhealthful when dissociated from the peel and pulp, as in many fruit juice drinks. When eaten as table sugar (50% of sucrose is fructose when digested), or high fructose corn syrup, it is devoid of all nutrients except calories. When I was a child in the '40s and '50s, orange juice was a rare treat and we were strictly limited to one 4 oz glass at most a couple of times per week. Today, people think nothing of drinking a 20 oz serving of fruit juice daily which contains 50-60 grams of sugar (~15 teaspoonsful), delicious fresh fruits are available year round, and high fructose corn syrup and/or sucrose are now used in the majority of commercial foods. "Sport" drinks didn't exist when I was young, and nobody was putting sugar in pizza or lasagna then, either. The rapid increase in high fructose corn syrup and fruit consumption during the '80s is now manifesting itself in poorer health for most. The point is that something in the American diet is screwing us up royally; and sugar consumption, and fructose in particular, appears to me to be the culprit. Actually, I wanted to put even more about the research in the article but haven't had time.

"''it's slower to digest therefore the pancreatic function is not overloaded''"
In actuality, fructose causes insulin resistance (a component of type II diabetes). This causes glucose levels to alway be higher than normal, increases the height of postprandial glucose peaks, and increases the glycation damage glucose causes. This is in addition to the seriously increased glycation activity of fructose, which sometimes predominates in cataracts. This is an indicator of lifetime glycation damage since the lens crystalline proteins are so long-lived.

"''because fructose (the sugar of fruits in most cases) has been arround humans and ancestors for million of years... the human body is very much acclimated to fructose consumption, and fructose can be considered for historical reasons to be the main physiological sugar for oral consumption."''

This statement is rather misleading. Glucose is the main sugar, and the primary fuel for our bodies. Fructose may be absent from the blood stream, but glucose is always there or you would be dead. Almost all fruits we consume today are modern cultivars with very much higher sugar content than the proto-fruits they were developed from and that we were evolved to eat. Think of it as crabapples vs Fuji appples. Any named varietal (Bartlett pears, Bing cherries, etc) is higher in sugar content than it's forebears. And, don't forget that 50% of sucrose is fructose. Free fructose is also found in fruits. Humans are adapted to eat fructose only in small quantities and it is rejected by the gut if glucose is not present.

*** I disagree about today's fruit having much higher sugar content. For example, the red delicious apple has been developed to look good... not necessarily to taste good. Fruit that is picked and eaten directly from the tree is also likely to be sweeter than fruit that's been sitting at the grocery store/factory for weeks. Finally, fruits in tropical countries are amazingly sweet and I would bet you that they were also plentiful and easily available. Think of mangoes and papayas, for instance. And if you really think about it, how much longer have fruit (fructose) been around compared to sugar from sugar cane or beets?
****

The lengthy metabolic pathway is one example; it is digested through the liver like a toxin. When fructose finally reaches the gluconeogenesis stage, it can go to glucose, glycogen, cholesterol, or lipids. It preferentially goes to cholesterol. Reduction of fructose consumption typically lowers blood cholesterol significantly with no other changes. My personal cholesterol went from 235 mg/dl to 180, a drop of about 50 points, in 18 months.

*** I would be curious if fructose consumption was all you reduced or if you reduced consumption of other foods as well. Also, since having a high HDL is actually good for you, I'm curious to know what your HDL/LDL ratio was before and after reduction of fructose, i.e. total cholesterol levels doesn't really tell us much.

The advent of high fructose corn syrup in the late '60s/early '70s as the lowest cost sweetener per dollar became a significant change in the American diet as it was widely implemented into commercial foods through the '80s. With the expected 15 year delay, for the results to start showing up, we now are seeing the detrimental health results of that increase in fructose consumption. Some public health experts are now concerned about a possible reduction in American average lifespan in spite of modern medical treatment advances.

A short list of recommended references:

1. Elliott SS, Keim NL, Stern JS, Teff K, Havel PJ. Fructose, weight gain,
and the insulin resistance syndrome. Am J Clin Nutr 2002;76:911–22.
[http://www.ajcn.org/cgi/content/full/76/5/911#SEC7]

2. International Obesity Task Force Website. August 2002: Internet:
http://www.iotf.org/media/syrup.htm .
3. Teff K, Elliott S, Tschoep M, et al. Consuming high fructose meals
reduces 24 hour plasma insulin and leptin concentrations, does not
suppress circulating ghrelin, and increases postprandial and fasting
triglycerides in women. Diabetes 2002;51(suppl):A408 (abstr).

4. Dills WL Jr. Protein fructosylation: fructose and the Maillard reaction.
Am J Clin Nutr 1993;58(suppl):779S–87S.

5. Bell RC, Carlson JC, Storr KC, Herbert K, Sivak J. High-fructose feeding
of streptozotocin-diabetic rats is associated with increased cataract formation
and increased oxidative stress in the kidney. Br J Nutr 2000;84:575–82.

6. Levi B, Werman MJ. Long-term fructose consumption accelerates
glycation and several age-related variables in male rats. J Nutr 1998;
128:1442–9.

7. Cohen JC, Schall R. Reassessing the effects of simple carbohydrates
on the serum triglyceride responses to fat meals. Am J Clin Nutr
1988;48:1031–4.

8. Crapo PA, Kolterman OG, Henry RR. Metabolic consequence of
two-week fructose feeding in diabetic subjects. Diabetes Care 1986;
9:111–9.

9. Bantle JP, Raatz SK, Thomas W, Georgopoulos A. Effects of dietary
fructose on plasma lipids in healthy subjects. Am J Clin Nutr 2000;
72:1128–34.

10. Abraha A, Humphreys SM, Clark ML, Matthews DR, Frayn KN.
Acute effect of fructose on postprandial lipaemia in diabetic and nondiabetic
subjects. Br J Nutr 1998;80:169–75.

11. Jeppesen J, Chen YI, Zhou MY, Schaaf P, Coulston A, Reaven GM.
Postprandial triglyceride and retinyl ester responses to oral fat: effects
of fructose. Am J Clin Nutr 1995;61:787–91.

12. Havel PJ, Elliott S, Keim NL, Krauss RM, Teff K. Short-term and
long-term consumption of high fructose, but not high glucose, diets
increases postprandial triglycerides and apo-lipoprotein-B in women.
J Invest Med 2003;52(suppl):S163 (abstr).

There are some traps in reading the literature:
1) glycosylation and glycation are often confused or used imprecisely. You must read syntax to determine what they really mean.
2) Corn syrup sometimes means high fructose corn syrup and sometimes plain old non-fructose corn syrup
3) Many early fructose-related studies used improper assay techniques for fructose vs glucose glycations and glycosylations that were off by up to several orders of magnitude. You must be aware which assay techniques are accurate and read the article to determine which were used. Also, often, no distinction between glucose glycations and fuctose glycations are made.
--[[User:Jwanderson|Jwanderson]] 07:30, 23 November 2005 (UTC)jwanderson
-----------------------------------------------

What do you mean that some people react badly to fructose??


==Full blown misguided views on health effects==

I added a NPOV warning for the following reasons :

Full blown misguided studies/articles about health effects :
Why would fructose cause obesity if it's slower to digest than sacharose and also more sweeter for less calories, and also les likely to drive any addiction, the study about obesity is absolutly ridiculous and should ne withdrawn from that page! And the other healt effect can be submited to the same considerations!

More considerations about health effect of fructose may be described as detrimental by the sugar industry because it makes huge profit on the addictive properties of glucose and sucrose that are often detrimental to the health because of poor mineral content, trigering insuline rush through hypo/hyper glycemia vicious cycles! In reallity fructose is much healtier than glucose and sucrose because :

- it's slower to digest therefore the pancreatic function is not overloaded, as a side effect it's very much tolerated by diabetic people!
- because it's often absorbed with fruits it's associated with many highly valuable nutriments!
- because fructose (the sugar of fruits in most cases) has been arround humans and ancestors for million of years (we know something about roots sugar just from industrial time) the human body is very much acclimated to fructose consumption, and fructose can be considered for historical reasons to be the main physiological sugar for oral consumption.
- nowdays we eat about 100 times more sugar than our ancestors 200 years ago!

We recall here that huge profit are made from addictive substances, sucrose/glucose can easily become addictive with many consequences : diabetis, tooth decay, probably hyper-activity and some burst of violence actually the main recommendation for glucose absorbtion is before an intense burst of physical activity! Not everyday for most people.


:Just because you don't agree with the article, it doesn't mean that it is biased. The linked study does not appear unscientific to me. If indeed a diet high in fructose leads to obesity in laboratory animals, and the same effect is considered likely in humans, then this is certainly worth mentioning in Wikipedia. --[[User:221.249.13.34|221.249.13.34]] 05:20, 22 September 2005 (UTC)

::Agree with previous poster, just because you don't agree doesn't mean it's not true! And as for that posted study, there actually was *another* one done with mice conducted by the University of Cincinnati just recently that apparently also supported the view that fructose for some reason fosters fat creation. Matter of fact it was all over the news here at one point. http://cincinnati.bizjournals.com/cincinnati/stories/2005/07/25/daily43.html <-is the first thing I've found about it. I actually with some effort managed to track down the article where they published their findings, but it's locked down behind one of those lame 'you need a subscription to view this article' sites. The study apparently compared mice drinking water, a sucrose sweetened commercial drink, and a mix of water and fructose. They apparently were rather suprised that both the soft drink and fructose water drinking mice actually ate less food than the mice with water, and thus the caloric intake ended up about the same. However, the fructose water mice gained a signifigant amount of weight versus the soft drink and water mice, apparently. From things I've read, they apparently also suggested that the recent obesity epidemic was caused by the introduction of 'high fructose corn syrup' to drinks. The industry didn't like that, and from what I've read I gather they attacked the study methods as well as the fact that the 'fructose water' used pure fructose, whereas they do not. That's two studies now that indiciate that fructose *by itself* anyway can cause increased fat creation versus normal sucrose. The best thing in this instance is to remember to step back and look at all sides. The article already mentions the effects with diabetics, and in facts states that it is 'hypothesized' that it *might* cause obesity. I really, really don't see any NPOV Problems with this article, tell the truth. And the article *also* mentions that fructose is generally found 'in combination with sucrose and glucose.' I've heard, though not personally seen, that studies have been done which show that the addition of glucose vastly reduced this effect. However, the article is about fructose itself, and as the views you oppose are presented as mere supposition, I don't understand how there's a NPOV Problem. -Graptor

:::I'm going to remove the NPOV dispute, since I think it's based on ignorance. Aside from the fact that there are many well-done studies on the relationship between fructose and obesity, the OP is clearly misinformed about fructose, since, in fact, it is the major sugar used in foods today (in the form of High Fructose Corn Syrup, HFC) by the "sugar industry" s/he lambasts. [[User:Graft|Graft]] 20:50, 30 September 2005 (UTC)

== Structures of L Forms ==
I have removed these structures from the article because they seem incorrect to me. They are not mirror images of the &alpha;-D and &beta;-D forms. The two "front-most" hydroxyls need to be "flipped". I think that the strucures shown are not fructose at all, but some other ketohexose.

[[Image:Alpha-L-Fructose-structure.png|thumb|Supposed &alpha;-L-Fructose]] [[Image:Beta-L-Fructose-structure.png|thumb|Supposed &beta;-L-Fructose]]

Oops, forgot to sign. [[User:Josh Cherry|Josh Cherry]] 00:23, 2 Jul 2004 (UTC)

I'm sorry about that, and taking a long time to fix it. I've updated the images, can they be included now? [[User:Sverdrup|{{User:Sverdrup/sig}}]] 08:16, 8 Sep 2004 (UTC)

They look right to me now. [[User:Josh Cherry|Josh Cherry]] 01:18, 9 Sep 2004 (UTC)

These graphics are still incorrect. In each graphic, where ever CH3 or H3C is found, it should be CH2 or H2C respectively. Thank you for placing these graphics online.... but they need to be accurate before they are borrowed and end up on websites and student papers all over the world. [[User: Don DeWitt]] 1 Nov 2004

The same problem exists for the D-isomers. I have created revised images in Photoshop but I do not know how to upload them. The original author may wish to make these corrections and obviously knows how to do it. [[User: Don DeWitt]] 1 Nov 2004

One last thought. It would be very helpful if the carbons were numbered in all the graphics. [[User: Don DeWitt]] 1 Nov 2004

=="Fructose is a blood sugar"?==
I'm a bit confused by the following sentence:
<blockquote>Fructose is a simple sugar (monosaccharide) found in many foods and '''one of the three most important blood sugars''' along with glucose and galactose.</blockquote>
According to the article about [[blood sugar]], the term ''blood sugar'' is only used to refer to the glycose levels in the blood. It's true that fructose and galactose can be converted into glycose, but does that really make them blood sugars themselves? If not, I think the above sentence should be rewritten, but if that's really the case, I would say the [[blood sugar]] article needs an update.
- [[User:Wintran|Wintran]] 03:54, 19 October 2005 (UTC)

:Erm... "blood sugar" as a medical term refers to glucose, which means glucose levels are used to meter the amount of sugar in the blood, but there are nevertheless other sugars present in the blood. [[User:Graft|Graft]] 04:09, 19 October 2005 (UTC)

Ah, I see. Do you think we can clarify this in the [[blood sugar]] article, that the term can also be used to refer to other types of less prominent sugars in the blood, and not just glucose? - [[User:Wintran|Wintran]] 12:20, 19 October 2005 (UTC)
:I'll try and clarify it. [[User:Graft|Graft]] 22:11, 20 October 2005 (UTC)
Thanks, looks much better now. - [[User:Wintran|Wintran]] 00:03, 22 October 2005 (UTC)

== Are Fructose and Levulose different? ==

This is in response to a statement in a [http://www.mercola.com/2006/feb/4/stop_marketing_sugary_cereals_to_kids.htm Mercola article] where he states <i>"CSPI has been on a campaign to lump all sugars together, claiming, wrongly, that corn-derived fructose is no different than the levulose that comes from fruit."</i> Any idea what he's getting on about? I thought fruits had fructose, not levulose, it'd make sense if they're the same but why use different terms... is the fructose in [[corn syrup]] not levulose? -[[User:Tyciol|Tyciol]] 10:06, 7 February 2006 (UTC)
:Uh. Possibly he's referring to the fact that corn syrup is mostly D-fructose whereas fruits are mostly L-fructose (levulose). I'm not even sure if this is true, it's just a hunch I have. Also I don't know whether this has any physiological implications. [[User:Graft|Graft]] 15:33, 7 February 2006 (UTC)

== Which form is the most common? ==

Can anyone tell me which form is the most common? I will then use that one as the base for an info table. [[User:RyanJones|Ryan Jones]] 20:09, 4 April 2006 (UTC)
:The quick answer is that the D isomer is the more common (natural) enantiomer. The tricky part is that it exists as an equillibrium mixture of open chain, pyranose and furanose forms - and each of the ring forms exist as mixtures of alpha and beta anomers. --[[User:Edgar181|Ed]] ([[User talk:Edgar181|Edgar181]]) 20:28, 4 April 2006 (UTC)

== Reference for more about metabolism ==

Removing the following from the article:

"
Articles

Carbohydrates and Increases in Obesity: Does the Type of Carbohydrate Make a Difference?
Judith Wylie-Rosett[http://www.obesityresearch.org/cgi/content/full/12/suppl_2/124S]

For more details about fructose metabolism see: * Horn, R., [http://www.medbio.info]
"

May be useful when expanding the article. - [[User:Samsara|{{{2|Samsara}}}]] ([[User talk:Samsara|talk]] • [[Special:Contributions/Samsara|contribs]]) 17:55, 22 April 2006 (UTC)

== Fructose for the hypoglycemic? ==

From the article:

:''Fructose is often recommended for, and consumed by, people with diabetes mellitus or '''hypoglycemia'''...''

Why would fructose be recommended for those with hypoglycemia? Shouldn't that be hyperglycemia? [[User:Cburnett|Cburnett]] 18:58, 12 June 2006 (UTC)


==Health effects ?==

I can't beleive that a natural ingredient of fruits be THAT bad for our health...
Would Fructose be supposedly bad because it is either "refined" like in white sugar, or manufactured like in HFCS, and therefore it lacks the necessary enzimes that might be present in unrefined sugar and fruits ?
[[User_talk:_dhrm77]]

:I agree, the health effects section seems very biased to me, with some unclear sources. - [[User:Wintran|Wintran]] <sup>([[User_talk:Wintran|talk]])</sup> 00:43, 31 July 2006 (UTC)

What I meant to say is :
Why is it that the fructose contained in fruits is good for us, but not the pure concentration that might be used as a sweetener ?
Is is a question of quantity ? A question of missing enzimes/minerals/vitamins ? or is it something else ?
[[User_talk:_dhrm77]]

:Well, because of the lack of neutrality in this article it feels to me like it draws the conclusion that fruits are bad, because fructose itself is bad. However, I agree with you that there are differences between fructose concentrations and natural fruits. For example, most (all?) fruits contain both glucose and fructose, so they aren't subject to the absorbation problems mentioned as a negative health effect.

:Regarding obesity problems it definitely seems more a quantity problem than quality. Overeating can be caused by any type of food and I've yet to see a research showing a clear connection between fructose and obesity that cannot be seen in other sugars that contain equal amounts of calories. - [[User:Wintran|Wintran]] <sup>([[User_talk:Wintran|talk]])</sup> 13:35, 31 July 2006 (UTC)

Latest revision as of 04:10, 30 August 2024

Presence in semen, and as proof in rape cases

[edit]

Semen is the only human product that contains any appreciable amount of fructose. For this reason it is a "marker" in rape cases. The logical place in Wikipedia for this information is in this article.38.104.70.30 (talk) 22:47, 24 February 2023 (UTC)captcrisis[reply]

Talk topic on contradictory information from EFSA's perspective?

[edit]

In Potential health effects section, there is a Self-contradictory template that states:

I'm guessing this is the mentioned talk post, which was archived back in Feb 2023, before the Mar 2023 mentioned in the template.

Should a new topic be started, or the existing one unarchived, or the template removed? As a casual user, I found it a bit confusing to figure out where to get more details, with there just being the one topic above that was left unarchived. But also not familiar enough with WP editing to know if it's normal to reference archived talk posts in the article. Pb7280 (talk) 23:10, 11 May 2023 (UTC)[reply]

The challenge from the March 2023 talk page about whether a controversy exists within the EFSA opinion was raised by an IP editor whose interpretation was incorrect. There is no controversy.
I removed the "contradicton box" with other minor edits, here. Zefr (talk) 00:19, 12 May 2023 (UTC)[reply]

Relative sweetness: weight or mole basis?

[edit]

It is important to say whether sweetness is compared on a molar or weight basis. If the basis is weight as I suspect, the enhanced sweetness of fructose over glucose could be explained by the fact that its molecular weight is about half that of sucrose. Hydrolysis of sucrose would have a smaller effect on sweetness, as only half the weight of sucrose is converted to fructose with greater sweetness, while the other half is converted to glucose with lesser sweetness.Eaberry (talk) 15:47, 18 January 2024 (UTC)[reply]

Did you mean 'sweetness of fructose over sucrose? That would be logical. However, the following reference says that fructose is twice as sweet on a molar basis:
by DT Cason · 1987 · Cited by 46 — Fructose is a very sweet sugar and on a molar basis is twice as sweet as sucrose ...
I haven't read enough to have confidence in that answer so I haven't edited the article. SciberDoc (talk) 15:08, 27 February 2024 (UTC)[reply]

Edit of table needed, please help if you have the skill required

[edit]

Table 1. Sugar content of selected common plant foods (g/100g The headings 'Fruits', 'Vegetables' and 'Grains' need deleting. They are inappropriate and in the wrong places. As the table can usefully be sorted, any division into fruits vegetables and grains is bound to go wrong depending which way it is sorted (unless each section sorted separately which would make comparisons harder between items in different categories).

I attempted to delete the headings but everything I tried broke the table display when I tried it in the sandbox. Help from someone skilled with tables would be appreciated. Thanks. SciberDoc (talk) 14:57, 27 February 2024 (UTC)[reply]

Absolute sweetness of fructose is identical at 5 °C as 50 °C?

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I was not able to find this information in the cited source (Lee, Thomas D. (1 January 2000). "Sweeteners". Kirk-Othmer Encyclopedia of Chemical Technology. doi:10.1002/0471238961.19230505120505.a01.pub2. ISBN 978-0471238966.). Does anyone know where did this information come from? 182.255.32.46 (talk) 04:04, 30 August 2024 (UTC)[reply]

Ok I found it, but in the "sugars" section, not "sweeteners". In fact I suspect that most of the citations of this source should be "sugars". The sweeteners part barely talked about fructose at all. 182.255.32.46 (talk) 04:10, 30 August 2024 (UTC)[reply]
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