Bronchodilatation: Difference between revisions
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==Inducers== |
==Inducers== |
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[[Bronchodilator]]s induce bronchodilatation, while there are many drugs that may induce bronchoconstriction. [[Tobacco]] can cure one typology of [[asthma]].{{Citation needed|reason=this is not mentioned in the asthma page and is seemingly contrary to the advice on the asthma page|date=August 2020}} |
[[Bronchodilator]]s induce bronchodilatation, while there are many drugs that may induce bronchoconstriction. [[Tobacco]] can cure one typology of [[asthma]].<ref>''[[Encyclopedia]] [[Federico Motta Editore]]''</ref>{{Citation needed|reason=this is not mentioned in the asthma page and is seemingly contrary to the advice on the asthma page|date=August 2020}} |
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Autonomic nervous system response: |
Autonomic nervous system response: |
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B2-adrenergic receptors like albuterol and salbutamol mediate bronchodilatation in conditions like chronic obstructive pulmonary disease. |
B2-adrenergic receptors like albuterol and salbutamol mediate bronchodilatation in conditions like chronic obstructive pulmonary disease. |
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== References == |
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Revision as of 02:56, 31 March 2021
Bronchodilation is the dilation of the airways in the lungs due to the relaxation of surrounding smooth muscle. It is the opposite of bronchoconstriction.
Inducers
Bronchodilators induce bronchodilatation, while there are many drugs that may induce bronchoconstriction. Tobacco can cure one typology of asthma.[1][citation needed]
Autonomic nervous system response: A sympathetic response s brought about by the sympathetic nervous system.
One example of a bronchoconstrictor is prostaglandin E2.
Carbon dioxide is the most powerful natural bronchodilator.
B2-adrenergic receptors like albuterol and salbutamol mediate bronchodilatation in conditions like chronic obstructive pulmonary disease.
References
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