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== Article Draft ==
== Article Draft ==


=== Lead ===
=== Overview ===
'''Follicular atresia''' is the breakdown of the [[ovarian follicles]] which are made in the ovaries and serve to produce hormones and oocytes. Oocytes are surrounded by [[granulosa cells]] and internal and external [[theca]] cells.<ref>{{Cite journal |last=Zhou |first=Jiawei |last2=Peng |first2=Xianwen |last3=Mei |first3=Shuqi |date=2019-01-29 |title=Autophagy in Ovarian Follicular Development and Atresia |url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6429023/ |journal=International Journal of Biological Sciences |volume=15 |issue=4 |pages=726–737 |doi=10.7150/ijbs.30369 |issn=1449-2288 |pmc=6429023 |pmid=30906205}}</ref>


=== Article body ===
=== Article body ===

Revision as of 22:34, 25 July 2022

It has been proposed that enhanced levels of Nitrogen oxide in rats can prevent atresia of the ovarian follicle, and depressed levels have the opposite effect.[1][2]

Article Draft

Overview

Follicular atresia is the breakdown of the ovarian follicles which are made in the ovaries and serve to produce hormones and oocytes. Oocytes are surrounded by granulosa cells and internal and external theca cells.[3]

Article body

References

  1. ^ Najati, Vahid; Ilkhanipour, Minoo; Salehi, Shahpar; Sadeghi-Hashjin, Goudarz (2008). "Role of nitric oxide on the generation of atretic follicles in the rat ovaries". Pakistan journal of biological sciences: PJBS. 11 (2): 250–254. doi:10.3923/pjbs.2008.250.254. ISSN 1028-8880. PMID 18817198.
  2. ^ Luo, Yuxin; Zhu, Yanbin; Basang, Wangdui; Wang, Xin; Li, Chunjin; Zhou, Xu (2021). "Roles of Nitric Oxide in the Regulation of Reproduction: A Review". Frontiers in Endocrinology. 12: 752410. doi:10.3389/fendo.2021.752410. ISSN 1664-2392. PMC 8640491. PMID 34867795.{{cite journal}}: CS1 maint: unflagged free DOI (link)
  3. ^ Zhou, Jiawei; Peng, Xianwen; Mei, Shuqi (2019-01-29). "Autophagy in Ovarian Follicular Development and Atresia". International Journal of Biological Sciences. 15 (4): 726–737. doi:10.7150/ijbs.30369. ISSN 1449-2288. PMC 6429023. PMID 30906205.