Thomas N. Sato: Difference between revisions
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'''Thomas N. Sato''' is a prominent Japanese educator and [[biologist]], whose research focuses on understanding molecular basis of [[cancer]], [[cardiac disease]] and [[metabolic diseases]] by using a number of [[animal models]] including [[mice]], [[zebrafish]] and [[fruit flies]]. He is also working to invent next-generation [[therapeutics]] for human diseases based on the stochastic basis of life and disease. He is currently [[ |
'''Thomas N. Sato''' is a prominent Japanese educator and [[biologist]], whose research focuses on understanding molecular basis of [[cancer]], [[cardiac disease]] and [[metabolic diseases]] by using a number of [[animal models]] including [[mice]], [[zebrafish]] and [[Drosophila melanogaster|fruit flies]]. He is also working to invent next-generation [[therapeutics]] for human diseases based on the stochastic basis of life and disease. He is currently [[professor]] of biology at the [[Nara Institute of Science and Technology]], Japan, professor at [[Cornell University]], United States and [[Centenary Institute]] in [[Sydney]], Australia.<ref>{{cite web|url=http://bsw3.naist.jp/tns/?cate=164&id=125 |title= Laboratory of Biodynamics and Integrative Biology|date= |accessdate=2013-07-23}}</ref><ref>{{cite web|url=http://bsw3.naist.jp/eng/courses/courses307.html |title= NAIST-Biological Science, Biodynamics and Integrative Biology|date= |accessdate=2013-07-23}}</ref> |
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==Life== |
==Life== |
Revision as of 19:03, 4 September 2013
Thomas N. Sato is a prominent Japanese educator and biologist, whose research focuses on understanding molecular basis of cancer, cardiac disease and metabolic diseases by using a number of animal models including mice, zebrafish and fruit flies. He is also working to invent next-generation therapeutics for human diseases based on the stochastic basis of life and disease. He is currently professor of biology at the Nara Institute of Science and Technology, Japan, professor at Cornell University, United States and Centenary Institute in Sydney, Australia.[1][2]
Life
- University of Tsukuba, Japan, BS 1985
- Georgetown University, Graduate School of Biological Sciences/Neuroscience, Ph.D. 1988
- Scripps Research Institute, Postdoctoral Fellow 1989-1990
- Roche Institute of Molecular Biology, Assistant Member 1991-1995
- Harvard Medical School, Assistant Professor 1995-1997
- Univ. of Texas Southwestern Medical School, Associate Professor 1997-2001 (Tenured in 1999)
- Univ. of Texas Southwestern Medical School, Professor 2002-2004
- Weill Medical College of Cornell University, Professor 2005-2006
- Weill Medical College of Cornell University, Joseph C. Hinsey Professor 2005-2009
- Nara Institute of Science and TechnologyGraduate School of Biological Sciences, Japan, Professor 2009–present
- Cornell University, Biomedical Engineering, Adjunct Professor 2009–present
- Centenary Institute, Sydney, Australia, Affiliate Professor 2009–present
Works
- Discoveries of Tie1, Tie2, and angiopoietins, that play the key roles in the blood vessel formation.[3][4][5]
References
- ^ "Laboratory of Biodynamics and Integrative Biology". Retrieved 2013-07-23.
- ^ "NAIST-Biological Science, Biodynamics and Integrative Biology". Retrieved 2013-07-23.
- ^ Sato TN; et al. (1993). "Tie-1 and tie-2 define another class of putative receptor tyrosine kinase genes expressed in early embryonic vascular system". Proc. Natl. Acad. Sci. USA. 90 (20): 9355–9358. PMID 8415706.
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ignored (help) - ^ Suri C, Jones PF, Patan S, Bartunkova S, Maisonpierre PC, Davis S, Sato TN, Yancopoulos GD (1996). "Requisite role of angiopoietin-1, a ligand for the TIE2 receptor, during embryonic angiogenesis". Cell. 87 (7): 1171–1180. doi:10.1016/S0092-8674(00)81813-9. PMID 8980224.
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ignored (help)CS1 maint: multiple names: authors list (link) - ^ Maisonpierre PC, Suri C, Jones PF, Bartunkova S, Wiegand SJ, Radziejewski C, Compton D, McClain J, Aldrich TH, Papadopoulos N, Daly TJ, Davis S, Sato TN, Yancopoulos GD (1997). "Angiopoietin-2, a Natural Antagonist for Tie2 That Disrupts in vivo Angiogenesis". Science. 277 (5322): 55–60. doi:10.1126/science.277.5322.55.
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ignored (help)CS1 maint: multiple names: authors list (link)