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====Eosinophilia====
====Eosinophilia====
{{Main|Eosinophilia}}
{{Main|Eosinophilia}}
When evaluating eosinophils it is important to evaluate the absolute numbers as opposed to the percentage, which depends on the total cell count and may be misleading. A normal eosinophil count is considered to be less than 0.65 x 10<sup>9</sup>/L.<ref>{{cite book|last=al.]|first=editors, Kenneth Kaushansky ... [et|title=Williams hematology|year=2010|publisher=McGraw-Hill Medical|location=New York|isbn=0071621512|edition=8th ed.}}</ref> Eosinophil counts are higher in newborns and vary with age, time (lower in the morning and higher at night), exercise, environment, and exposure to allergens.<ref>{{cite book|last=al.]|first=editors, Kenneth Kaushansky ... [et|title=Williams hematology|year=2010|publisher=McGraw-Hill Medical|location=New York|isbn=0071621512|edition=8th ed.}}</ref> [[Beta blocker|Beta blockers]] can also cause an increase in the eosinophil count.<ref>{{cite book|last=al.]|first=editors, Kenneth Kaushansky ... [et|title=Williams hematology|year=2010|publisher=McGraw-Hill Medical|location=New York|isbn=0071621512|edition=8th ed.}}</ref>
When evaluating eosinophils it is important to use the absolute numbers as opposed to the percentage. Percentages depend on the total cell count and may be misleading. A normal eosinophil count is considered to be less than 0.65 x 10<sup>9</sup>/L.<ref>{{cite book|last=al.]|first=editors, Kenneth Kaushansky ... [et|title=Williams hematology|year=2010|publisher=McGraw-Hill Medical|location=New York|isbn=0071621512|edition=8th ed.}}</ref> Eosinophil counts are higher in newborns and vary with age, time (lower in the morning and higher at night), exercise, environment, and exposure to allergens.<ref>{{cite book|last=al.]|first=editors, Kenneth Kaushansky ... [et|title=Williams hematology|year=2010|publisher=McGraw-Hill Medical|location=New York|isbn=0071621512|edition=8th ed.}}</ref> [[Beta blocker|Beta blockers]] can also cause an increase in the eosinophil count.<ref>{{cite book|last=al.]|first=editors, Kenneth Kaushansky ... [et|title=Williams hematology|year=2010|publisher=McGraw-Hill Medical|location=New York|isbn=0071621512|edition=8th ed.}}</ref>

Revision as of 17:03, 11 December 2013

Proliferative disorders

Main article: Leukocytosis

An increase in the number of white blood cells in circulation is called leukocytosis.[1] This increase is most commonly caused by inflammation.[1] There are four major causes: increase of production in bone marrow, increased release from storage in bone marrow, decreased attachment to veins and arteries, decreased uptake by tissues.[1] Leukocytosis may affect one or more cell lines and can be neutrophilic, eosinophilic, basophilic, monocytosis, or lymphocytosis.

Neutrophilia

Main article: Neutrophilia

Neutrophilia is an increase in the absolute neutrophil count in the peripheral circulation. Normal blood values vary by age.[2] Neutrophilia can be caused by a direct problem with blood cells (primary disease). It can also occur as a consequence of an underlying disease (secondary). Most cases of neutrophilia are secondary to inflammation.[3]

Primary Causes[4]

Secondary Causes[5]

Eosinophilia

Main article: Eosinophilia

When evaluating eosinophils it is important to use the absolute numbers as opposed to the percentage. Percentages depend on the total cell count and may be misleading. A normal eosinophil count is considered to be less than 0.65 x 109/L.[6] Eosinophil counts are higher in newborns and vary with age, time (lower in the morning and higher at night), exercise, environment, and exposure to allergens.[7] Beta blockers can also cause an increase in the eosinophil count.[8]

  1. ^ a b c Cite error: The named reference Al2010 was invoked but never defined (see the help page).
  2. ^ Cite error: The named reference Al2010a was invoked but never defined (see the help page).
  3. ^ Cite error: The named reference Schafer was invoked but never defined (see the help page).
  4. ^ Schafer, [edited by] Lee Goldman, Andrew I. Goldman's Cecil medicine (24th ed. ed.). Philadelphia: Elsevier/Saunders. ISBN 1437716040. {{cite book}}: |edition= has extra text (help); |first= has generic name (help)CS1 maint: multiple names: authors list (link)
  5. ^ Schafer, [edited by] Lee Goldman, Andrew I. Goldman's Cecil medicine (24th ed. ed.). Philadelphia: Elsevier/Saunders. ISBN 1437716040. {{cite book}}: |edition= has extra text (help); |first= has generic name (help)CS1 maint: multiple names: authors list (link)
  6. ^ al.], editors, Kenneth Kaushansky ... [et (2010). Williams hematology (8th ed. ed.). New York: McGraw-Hill Medical. ISBN 0071621512. {{cite book}}: |edition= has extra text (help); |first= has generic name (help)CS1 maint: multiple names: authors list (link)
  7. ^ al.], editors, Kenneth Kaushansky ... [et (2010). Williams hematology (8th ed. ed.). New York: McGraw-Hill Medical. ISBN 0071621512. {{cite book}}: |edition= has extra text (help); |first= has generic name (help)CS1 maint: multiple names: authors list (link)
  8. ^ al.], editors, Kenneth Kaushansky ... [et (2010). Williams hematology (8th ed. ed.). New York: McGraw-Hill Medical. ISBN 0071621512. {{cite book}}: |edition= has extra text (help); |first= has generic name (help)CS1 maint: multiple names: authors list (link)
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