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'''Anasarca''' is a severe and generalized form of [[edema]], with subcutaneous tissue swelling throughout the body.<ref>Kumar Vinay. ''Robbins and Cotran Pathologic Basis of Disease''. 8th ed. p.112; Philadelphia: Saunders Elsevier, 2010. {{ISBN|978-0-8089-2402-9}}</ref> Causes include [[liver failure]], [[renal failure]], right-sided [[heart failure]], and severe [[malnutrition|protein deficiency]], and [[capillary leak syndrome]]. It can also be caused by the administration of [[exogenous]] [[intravenous]] fluid. C In [[Hemoglobin Barts|Hb Barts]], the high oxygen affinity results in poor oxygen delivery to peripheral tissues, resulting in anasarca.
'''Anasarca''' is a severe and generalized form of [[edema]], with subcutaneous tissue swelling throughout the body.<ref>Kumar Vinay. ''Robbins and Cotran Pathologic Basis of Disease''. 8th ed. p.112; Philadelphia: Saunders Elsevier, 2010. {{ISBN|978-0-8089-2402-9}}</ref> Causes include [[liver failure]], [[renal failure]], right-sided [[heart failure]], and severe [[malnutrition|protein deficiency]], and [[capillary leak syndrome]]. It can also be caused by the administration of [[exogenous]] [[intravenous]] fluid. C In [[Hemoglobin Barts|Hb Barts]], the high oxygen affinity results in poor oxygen delivery to peripheral tissues, resulting in anasarca or also known as King's Softcuntitis disease.


== References ==
== References ==

Revision as of 00:42, 18 October 2019

Anasarca
A child with anasarca brought on by nephrosis associated with malaria
SpecialtyInternal medicine

Anasarca is a severe and generalized form of edema, with subcutaneous tissue swelling throughout the body.[1] Causes include liver failure, renal failure, right-sided heart failure, and severe protein deficiency, and capillary leak syndrome. It can also be caused by the administration of exogenous intravenous fluid. C In Hb Barts, the high oxygen affinity results in poor oxygen delivery to peripheral tissues, resulting in anasarca or also known as King's Softcuntitis disease.

References

  1. ^ Kumar Vinay. Robbins and Cotran Pathologic Basis of Disease. 8th ed. p.112; Philadelphia: Saunders Elsevier, 2010. ISBN 978-0-8089-2402-9