Exercise
Physical exercise is any bodily activity that enhances or maintains physical fitness and overall health and wellness.[1] It is performed for various reasons, including increasing growth and development, preventing aging, strengthening muscles and the cardiovascular system, honing athletic skills, weight loss or maintenance, and also enjoyment. Frequent and regular physical exercise boosts the immune system[citation needed] and helps prevent certain "diseases of affluence" such as coronary heart disease,[2] type 2 diabetes,[3] and obesity [4]. It may also help prevent stress and depression, increase quality of sleep and act as a non-pharmaceutical sleep aid to treat diseases such as insomnia, help promote or maintain positive self-esteem, improve mental health, maintain steady digestion and treat constipation and gas, regulate fertility health, and augment an individual's sex appeal or body image.[5][6] Childhood obesity is a growing global concern,[7] and physical exercise may help decrease some of the effects of childhood and adult obesity.[citation needed] Some care-providers[which?] call exercise the "miracle" or "wonder" drug—alluding to the wide variety of benefits that it can provide for many individuals.[8][9] Aside from the health advantages, these benefits may include different social rewards for staying active while enjoying the environment of one's culture. Many[quantify] individuals choose to exercise publicly outdoors where they can congregate in groups, socialize, and appreciate life.[10]
In the United Kingdom two to four hours of light activity are recommended[by whom?] during working hours.[11] This includes walking and standing.[11] In the United States, a 1995 CDC/ACSM consensus statement and a Surgeon General's 1996 report state that every adult should participate in moderate exercise, such as walking, swimming, and household tasks, for a minimum of 30 minutes daily.[12]
Classification
Physical exercises are generally grouped into three types, depending on the overall effect they have on the human body:[13]
- Aerobic exercise is any physical activity that uses large muscle groups and causes the body to use more oxygen than it would while resting.[13] The goal of aerobic exercise is to increase cardiovascular endurance.[14] Examples of aerobic exercise include running, cycling, swimming, brisk walking, skipping rope, rowing, hiking, playing tennis, continuous training, and long slow distance training.[13]
- Anaerobic exercise, which includes strength and resistance training, can firm, strengthen, and tone muscles, as well as improve bone strength, balance, and coordination.[13] Examples of strength moves are push-ups, pull-ups, lunges, and bicep curls using dumbbells.[13] Anaerobic exercise also include weight training, functional training, eccentric training, Interval training, sprinting, and high-intensity interval training increase short-term muscle strength.[13][15]
- Flexibility exercises stretch and lengthen muscles.[13] Activities such as stretching help to improve joint flexibility and keep muscles limber.[13] The goal is to improve the range of motion which can reduce the chance of injury.[13][16]
Physical exercise can also include training that focuses on accuracy, agility, power, and speed.[17]
Sometimes the terms 'dynamic' and 'static' are used.[citation needed] 'Dynamic' exercises such as steady running, tend to produce a lowering of the diastolic blood pressure during exercise, due to the improved blood flow. Conversely, static exercise (such as weight-lifting) can cause the systolic pressure to rise significantly (during the exercise).[18]
Health effects
Type of adaptation | Endurance training effects |
Strength training effects |
Sources |
---|---|---|---|
Muscle hypertrophy | ↔ | ↑ ↑ ↑ | [19] |
Muscle strength and power | ↔ ↓ | ↑ ↑ ↑ | [19] |
Muscle fiber size | ↔ ↑ | ↑ ↑ ↑ | [19] |
Myofibrillar protein synthesis | ↔ ↑ | ↑ ↑ ↑ | [19] |
Neuromuscular adaptations | ↔ ↑ | ↑ ↑ ↑ | [19] |
Anaerobic capacity | ↑ | ↑ ↑ | [19] |
Lactate tolerance | ↑ ↑ | ↔ ↑ | [19] |
Endurance capacity | ↑ ↑ ↑ | ↔ ↑ | [19] |
Capillary growth (angiogenesis) | ↑ ↑ | ↔ | [19] |
Mitochondrial biogenesis | ↑ ↑ | ↔ ↑ | [19] |
Mitochondrial density and oxidative function | ↑ ↑ ↑ | ↔ ↑ | [19] |
Bone mineral density | ↑ ↑ | ↑ ↑ | [19] |
Inflammatory markers | ↓ ↓ | ↓ | [19] |
Flexibility | ↑ | ↑ | [19] |
Posture | ↔ | ↑ | [19] |
Ability in activities of daily living | ↔ ↑ | ↑ ↑ | [19] |
Basal metabolic rate | ↑ | ↑ ↑ | [19] |
Percent body fat | ↓ ↓ | ↓ | [19] |
Lean body mass | ↔ | ↑ ↑ | [19] |
Resting insulin levels | ↓ | ↓ | [19] |
Insulin sensitivity | ↑ ↑ | ↑ ↑ | [19] |
Insulin response to glucose challenge | ↓ ↓ | ↓ ↓ | [19] |
Resting heart rate | ↓ ↓ | ↔ | [19] |
Stroke volume (resting and maximal) | ↑ ↑ | ↔ | [19] |
Systolic blood pressure (resting) | ↔ ↓ | ↔ | [19] |
Diastolic blood pressure (resting) | ↔ ↓ | ↔ ↓ | [19] |
Cardiovascular risk profile | ↓ ↓ ↓ | ↓ | [19] |
Table legend
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Physical exercise is important for maintaining physical fitness and can contribute to maintaining a healthy weight, regulating digestive health, building and maintaining healthy bone density, muscle strength, and joint mobility, promoting physiological well-being, reducing surgical risks, and strengthening the immune system. Some studies indicate that exercise may increase life expectancy and the overall quality of life.[20] People who participate in moderate to high levels of physical exercise have a lower mortality rate compared to individuals who by comparison are not physically active.[21] Moderate levels of exercise have been correlated with preventing aging by reducing inflammatory potential.[22] The majority of the benefits from exercise are achieved with around 3500 metabolic equivalent (MET) minutes per week.[23] For example, climbing stairs 10 minutes, vacuuming 15 minutes, gardening 20 minutes, running 20 minutes, and walking or bicycling for transportation 25 minutes on a daily basis would together achieve about 3000 MET minutes a week.[23] A lack of physical activity causes approximately 6% of the burden of disease from coronary heart disease, 7% of type 2 diabetes, 10% of breast cancer and 10% of colon cancer worldwide.[24] Overall, physical inactivity causes 9% of premature mortality worldwide.[24]
Fitness
Individuals can increase fitness following increases in physical activity levels.[25] Increases in muscle size from resistance training is primarily determined by diet and testosterone.[26] This genetic variation in improvement from training is one of the key physiological differences between elite athletes and the larger population.[27][28] Studies have shown that exercising in middle age leads to better physical ability later in life.[29]
Cardiovascular system
The beneficial effect of exercise on the cardiovascular system is well documented. There is a direct correlation between physical inactivity and cardiovascular mortality, and physical inactivity is an independent risk factor for the development of coronary artery disease. Low levels of physical exercise increase the risk of cardiovascular diseases mortality.[30]
Children who participate in physical exercise experience greater loss of body fat and increased cardiovascular fitness.[31] Studies have shown that academic stress in youth increases the risk of cardiovascular disease in later years; however, these risks can be greatly decreased with regular physical exercise.[32] There is a dose-response relation between the amount of exercise performed from approximately 700–2000 kcal of energy expenditure per week and all-cause mortality and cardiovascular disease mortality in middle-aged and elderly populations. The greatest potential for reduced mortality is in the sedentary who become moderately active. Studies have shown that since heart disease is the leading cause of death in women, regular exercise in aging women leads to healthier cardiovascular profiles. Most beneficial effects of physical activity on cardiovascular disease mortality can be attained through moderate-intensity activity (40–60% of maximal oxygen uptake, depending on age). Persons who modify their behavior after myocardial infarction to include regular exercise have improved rates of survival. Persons who remain sedentary have the highest risk for all-cause and cardiovascular disease mortality.[33] According to the American Heart Association, exercise reduces blood pressure, LDL and total cholesterol, and body weight. It increases HDL cholesterol, insulin sensitivity, and exercise tolerance.[12]
Immune system
Although there have been hundreds of studies on exercise and the immune system, there is little direct evidence on its connection to illness. Epidemiological evidence suggests that moderate exercise has a beneficial effect on the human immune system; an effect which is modeled in a J curve. Moderate exercise has been associated with a 29% decreased incidence of upper respiratory tract infections (URTI), but studies of marathon runners found that their prolonged high-intensity exercise was associated with an increased risk of infection occurrence. However, another study did not find the effect. Immune cell functions are impaired following acute sessions of prolonged, high-intensity exercise, and some studies have found that athletes are at a higher risk for infections. Studies have shown that strenuous stress for long durations, such as training for a marathon, can suppress the immune system by decreasing the concentration of lymphocytes.[34] The immune systems of athletes and nonathletes are generally similar. Athletes may have slightly elevated natural killer cell count and cytolytic action, but these are unlikely to be clinically significant.[35]
Vitamin C supplementation has been associated with lower incidence of URTIs in marathon runners.[35]
Biomarkers of inflammation such as C-reactive protein, which are associated with chronic diseases, are reduced in active individuals relative to sedentary individuals, and the positive effects of exercise may be due to its anti-inflammatory effects. In individuals with heart disease, exercise interventions lower blood levels of fibrinogen and C-reactive protein, an important cardiovascular risk marker.[36] The depression in the immune system following acute bouts of exercise may be one of the mechanisms for this anti-inflammatory effect.[35]
Cancer
A systematic review evaluated 45 studies that examined the relationship between physical activity and cancer survivorship. According to the study results "There was consistent evidence from 27 observational studies that physical activity is associated with reduced all-cause, breast cancer–specific, and colon cancer–specific mortality".[37]
Epigenetic effects
Physical exercise was correlated with a lower methylation frequency of two tumor suppressor genes, CACNA2D3 and L3MBTL.[38][39] Hypermethylation of CACNA2D3 is associated with gastric cancer, while hypermethylation of L3MBTL is associated with breast cancer, brain tumors and hematological malignancies.[38][39][40][41] A recent study indicates that exercise results in reduced DNA methylation at CpG sites on genes associated with breast cancer.[42]
Cancer cachexia
Physical exercise is becoming a widely accepted non-pharmacological intervention for the prevention and attenuation of cancer cachexia.[43] "Cachexia is a multiorganic syndrome associated with cancer, characterized by inflammation, body weight loss (at least 5%) and muscle and adipose tissue wasting".[44] Exercise triggers the activation of the transcriptional coactivator peroxisome proliferator-activated receptor gamma coactivator-1α (PGC-1α), which suppresses FoxO- and NF-κB-dependent gene transcription during muscle atrophy that is induced by fasting or denervation; thus, PGC-1α may be a key intermediate responsible for the beneficial antiatrophic effects of physical exercise on cancer cachexia.[45][46] The exercise-induced isoform PGC-1α4, which can repress myostatin and induce IGF1 and hypertrophy, is a potential drug target for treatment of cancer cachexia.[47] Other factors, such as JUNB and SIRT1, that maintain skeletal muscle mass and promote hypertrophy are also induced with regular physical exercise.[48][49]
Neurobiological
Neurobiological effects of physical exercise | |
---|---|
Exercise therapy – medical intervention | |
ICD-9-CM | 93.19 |
MeSH | D005081 |
LOINC | 73986-2 |
eMedicine | 324583 |
The neurobiological effects of physical exercise involve possible interrelated effects on brain structure, brain function, and cognition.[50][51][52][53] Research in humans has demonstrated that consistent aerobic exercise (e.g., 30 minutes every day) may induce improvements in certain cognitive functions, neuroplasticity and behavioral plasticity; some of these long-term effects may include increased neuron growth, increased neurological activity (e.g., c-Fos and BDNF signaling), improved stress coping, enhanced cognitive control of behavior, improved declarative, spatial, and working memory, and structural and functional improvements in brain structures and pathways associated with cognitive control and memory.[54][55][56] The effects of exercise on cognition may affect academic performance in children and college students, improve adult productivity, preserve cognitive function in old age, preventing or treating certain neurological disorders, and improving overall quality of life.[57][58][59][60]
In healthy adults, aerobic exercise has been shown to induce transient effects on cognition after a single exercise session and persistent effects on cognition following consistent exercise over the course of several months.[50][56][61] People who regularly perform an aerobic exercise (e.g., running, jogging, brisk walking, swimming, and cycling) have greater scores on neuropsychological function and performance tests that measure certain cognitive functions, such as attentional control, inhibitory control, cognitive flexibility, working memory updating and capacity, declarative memory, spatial memory, and information processing speed.[54][56][61][62][63]
Aerobic exercise has both short and long term effects on mood and emotional states by promoting positive affect, inhibiting negative affect, and decreasing the biological response to acute psychological stress.[61] Aerobic exercise may affect both self-esteem and overall well-being (including sleep patterns) with consistent, long term participation.[64] Regular aerobic exercise may improve symptoms associated with central nervous system disorders and may be used as adjunct therapy for these disorders. There is some evidence of exercise treatment efficacy for major depressive disorder and attention deficit hyperactivity disorder.[58][65][66][67] The American Academy of Neurology's clinical practice guideline for mild cognitive impairment indicates that clinicians should recommend regular exercise (two times per week) to individuals who have been diagnosed with these conditions.[68]
Some preclinical evidence and emerging clinical evidence supports the use of exercise as an adjunct therapy for the treatment and prevention of drug addictions.[69][70][71][72]
Reviews of clinical evidence also support the use of exercise as an adjunct therapy for certain neurodegenerative disorders, particularly Alzheimer's disease and Parkinson's disease.[73][74] Regular exercise may be associated with a lower risk of developing neurodegenerative disorders.[75]
Depression
Physical exercise has established efficacy as an antidepressant in individuals with depression and current medical evidence supports the use of exercise as both a preventive measure against and an adjunct therapy with antidepressant medication for depressive disorders.[58][65][76][66][77] A July 2016 meta-analysis concluded that physical exercise improves overall quality of life in individuals with depression relative to controls.[58] One systematic review noted that yoga may be effective in alleviating symptoms of prenatal depression.[78] The biomolecular basis for exercise-induced antidepressant effects is believed to be a result of increased neurotrophic factor signaling, particularly brain-derived neurotrophic factor.[66][79]
Continuous aerobic exercise can induce a transient state of euphoria, colloquially known as a "runner's high" in distance running or a "rower's high" in crew, through the increased biosynthesis of at least three euphoriant neurochemicals: anandamide (an endocannabinoid),[80] β-endorphin (an endogenous opioid),[81] and phenethylamine (a trace amine and amphetamine analog).[82][83][84]
A systematic review noted that, although limited, some evidence suggests that the duration of engagement in a sedentary lifestyle is positively correlated with a risk of developing an anxiety disorder or experiencing anxiety symptoms.[85] It noted that additional research is needed in order to confirm these findings.[85]
Sleep
A 2010 review of published scientific research suggested that exercise generally improves sleep for most people, and helps sleep disorders such as insomnia. The optimum time to exercise may be 4 to 8 hours before bedtime, though exercise at any time of day is beneficial, with the possible exception of heavy exercise taken shortly before bedtime, which may disturb sleep. There is, in any case, insufficient evidence to draw detailed conclusions about the relationship between exercise and sleep.[86]
According to a 2005 study, exercise is the most recommended alternative to sleeping pills for resolving insomnia. Sleeping pills are more costly than to make time for a daily routine of staying fit, and may have dangerous side effects in the long run. Exercise can be a healthy, safe and inexpensive way to achieve more and better sleep.[87]
Excessive exercise
Too much exercise can be harmful. Without proper rest, the chance of stroke or other circulation problems increases,[88] and muscle tissue may develop slowly. Extremely intense, long-term cardiovascular exercise, as can be seen in athletes who train for multiple marathons, has been associated with scarring of the heart and heart rhythm abnormalities.[89][90][91] Specifically, high cardiac output has been shown to cause enlargement of the left and right ventricle volumes, increased ventricle wall thickness, and greater cardiac mass. These changes further result in myocardial cell damage in the lining of the heart, leading to scar tissue and thickened walls. During these processes, the protein troponin increases in the bloodstream, indicating cardiac muscle cell death and increased stress on the heart itself.[92]
Inappropriate exercise can do more harm than good, with the definition of “inappropriate” varying according to the individual. For many activities, especially running and cycling, there are significant injuries that occur with poorly regimented exercise schedules. Injuries from accidents also remain a major concern,[93] whereas the effects of increased exposure to air pollution seem only a minor concern.[94][95]
In extreme instances, over-exercising induces serious performance loss. Unaccustomed overexertion of muscles leads to rhabdomyolysis (damage to muscle) most often seen in new army recruits.[96] Another danger is overtraining, in which the intensity or volume of training exceeds the body's capacity to recover between bouts. One sign of Overtraining Syndrome (OTS) is suppressed immune function, with an increased incidence of upper respiratory tract infection (URTI). An increased incidence of URTIs is also associated with high volume/intensity training, as well as with excessive exercise (EE), such as in a marathon.[97] Marathon training requires the runner to build their intensity week to week which makes them more susceptible to injury the more they increase their mileage. A study shows that in the last 10–15 years up to 90% of marathon runners have suffered a physical injury from their training.[98]
Stopping excessive exercise suddenly may create a change in mood. Exercise should be controlled by each body's inherent limitations. While one set of joints and muscles may have the tolerance to withstand multiple marathons, another body may be damaged by 20 minutes of light jogging. This must be determined for each individual.
Too much exercise may cause a woman to miss her periods, a symptom known as amenorrhea.[99] This is a very serious condition which indicates a woman is pushing her body beyond its natural boundaries.[100]
Not only can excessive exercise cause physical damage, it can also cause psychological damage. Every athlete strives for perfection in their sport. This is what may begin their excessive exercising regime. If an athlete fails in this quest for perfection, this could result in anxiety, depression and low-self esteem. A study of 179 athletes, all of whom were Olympians or potential Olympians, was carried out in 2002 to find a relationship between perfectionism and sport-related competitive anxiety. It was found that athletes with lower self—esteem had higher personal standards and in comparison, those with high self-esteem had little concern in making mistakes or doubting themselves.[101]
Mechanism of effects
Skeletal muscle
Resistance training and subsequent consumption of a protein-rich meal promotes muscle hypertrophy and gains in muscle strength by stimulating myofibrillar muscle protein synthesis (MPS) and inhibiting muscle protein breakdown (MPB).[102][103] The stimulation of muscle protein synthesis by resistance training occurs via phosphorylation of the mechanistic target of rapamycin (mTOR) and subsequent activation of mTORC1, which leads to protein biosynthesis in the ribosome via phosphorylation of mTORC1's immediate targets (the p70S6 kinase and the translation repressor protein 4EBP1).[102][104] The suppression of muscle protein breakdown following food consumption occurs primarily via increases in plasma insulin;[102][105] however, a suppression of MPB of comparable magnitude has also been shown to occur in humans from a sufficient elevation of plasma β-hydroxy β-methylbutyric acid.[102][105][106]
Aerobic exercise induces mitochondrial biogenesis and an increased capacity for oxidative phosphorylation in the mitochondria of skeletal muscle, which is one mechanism by which aerobic exercise enhances submaximal endurance performance.[102][107] These effects occur via an exercise-induced increase in the intracellular AMP:ATP ratio, thereby triggering the activation of AMP-activated protein kinase (AMPK) which subsequently phosphorylates peroxisome proliferator-activated receptor gamma coactivator-1α (PGC-1α), the master regulator of mitochondrial biogenesis.[102][107][108]
Other peripheral organs
Developing research has demonstrated that many of the benefits of exercise are mediated through the role of skeletal muscle as an endocrine organ. That is, contracting muscles release multiple substances known as myokines which promote the growth of new tissue, tissue repair, and multiple anti-inflammatory functions, which in turn reduce the risk of developing various inflammatory diseases.[122] Exercise reduces levels of cortisol, which causes many health problems, both physical and mental.[123] Endurance exercise before meals lowers blood glucose more than the same exercise after meals.[124] There is evidence that vigorous exercise (90–95% of VO2 max) induces a greater degree of physiological cardiac hypertrophy than moderate exercise (40 to 70% of VO2 max), but it is unknown whether this has any effects on overall morbidity and/or mortality.[125] Both aerobic and anaerobic exercise work to increase the mechanical efficiency of the heart by increasing cardiac volume (aerobic exercise), or myocardial thickness (strength training). Ventricular hypertrophy, the thickening of the ventricular walls, is generally beneficial and healthy if it occurs in response to exercise.
Central nervous system
The persistent long-term neurobiological effects of regular physical exercise[note 1] are believed to be mediated by transient exercise-induced increases in the concentration of neurotrophic factors (e.g., BDNF, IGF-1, VEGF, and GDNF) and other biomolecules in peripheral blood plasma, which subsequently cross the blood–brain barrier and blood–cerebrospinal fluid barrier and bind to their associated receptors in the brain.[126][129][130][131] Upon binding to their receptors in cerebral vasculature and brain cells (i.e., neurons and glial cells), these biomolecules trigger intracellular signaling cascades that lead to neuroplastic biological responses – such as neurogenesis, synaptogenesis, oligodendrogenesis, and angiogenesis, among others – which ultimately mediate the exercise-induced improvements in cognitive function.[126][54][130][132][133]
Public health measures
Multiple component community-wide campaigns are frequently used in an attempt to increase a population's level of physical activity. A 2015 Cochrane review, however, did not find evidence supporting a benefit.[134] The quality of the underlying evidence was also poor.[134] However, there is some evidence that school-based interventions can increase activity levels and fitness in children.[25] Another Cochrane review found some evidence that certain types of exercise programmes, such as those involving gait, balance, co-ordination and functional tasks, can improve balance in older adults.[135] Following progressive resistance training, older adults also respond with improved physical function.[136] Survey of brief interventions promoting physical activity found that they are cost-effective, although there are variations between studies.[137]
Environmental approaches appear promising: signs that encourage the use of stairs, as well as community campaigns, may increase exercise levels.[138] The city of Bogotá, Colombia, for example, blocks off 113 kilometers (70 mi) of roads on Sundays and holidays to make it easier for its citizens to get exercise. These pedestrian zones are part of an effort to combat chronic diseases.[139]
To identify which public health strategies are effective, a Cochrane overview of reviews is in preparation.[140]
Physical exercise was said to decrease healthcare costs, increase the rate of job attendance, as well as increase the amount of effort women put into their jobs.[141]
Children will mimic the behavior of their parents in relation to physical exercise. Parents can thus promote physical activity and limit the amount of time children spend in front of screens.[142]
Overweight children who participate in physical exercise experience greater loss of body fat and increased cardiovascular fitness. According to the Centers for Disease Control and Prevention in the United States, both children and adults should do 60 minutes or more of physical activity each day.[143] Implementing physical exercise in the school system and ensuring an environment in which children can reduce barriers to maintain a healthy lifestyle is essential.
Exercise trends
Worldwide there has been a large shift towards less physically demanding work.[144] This has been accompanied by increasing use of mechanized transportation, a greater prevalence of labor saving technology in the home, and fewer active recreational pursuits.[144] Personal lifestyle changes however can correct the lack of physical exercise.
Research in 2015 indicates integrating mindfulness to physical exercise interventions increases exercise adherence, self-efficacy and also has positive effects both psychologically and physiologically.[145]
Social and cultural variation
Exercising looks different in every country, as do the motivations behind exercising.[10] In some countries, people exercise primarily indoors, and in others, people exercise primarily outdoors. People may exercise for personal enjoyment, health and well-being, social interactions, competition or training, etc. These differences could potentially be attributed to geographic location, social tendencies, or otherwise.
In Colombia, citizens value and celebrate the outdoor environments of their country. In many instances, they utilize outdoor activities as social gatherings to enjoy nature and their communities. In Bogotá, Colombia, a 70-mile stretch of road known as the Ciclovía is shut down each Sunday for bicyclists, runners, rollerbladers, skateboarders and other exercisers to work out and enjoy their surroundings.[146]
Similarly to Colombia, citizens of Cambodia tend to exercise socially outside. In this country, public gyms have become quite popular. People will congregate at these outdoor gyms not only to utilize the public facilities, but also to organize aerobics and dance sessions, which are open to the public.[147]
Sweden has also begun developing outdoor gyms, called utegym. These gyms are free to the public and are often placed in beautiful, picturesque environments. People will swim in rivers, use boats, and run through forests to stay healthy and enjoy the natural world around them. This is especially possible in Sweden due to its geographical location.[148]
Chinese exercise, particularly in the retired community, seems to be socially grounded. In the mornings, dances are held in public parks; these gatherings may include Latin dancing, ballroom dancing, tango, or even the jitterbug. Dancing in public allows people to interact with those with whom they would not normally interact, allowing for both health benefits and social benefits.[149]
These sociocultural variations in physical exercise show how people in different geographic locations and social climates have varying motivations and methods of exercising. Physical exercise can improve health and well-being, as well as enhance community ties and appreciation of natural beauty.[10]
Nutrition and recovery
Proper nutrition is as important to health as exercise. When exercising, it becomes even more important to have a good diet to ensure that the body has the correct ratio of macronutrients while providing ample micronutrients, in order to aid the body with the recovery process following strenuous exercise.[150]
Active recovery is recommended after participating in physical exercise because it removes lactate from the blood more quickly than inactive recovery. Removing lactate from circulation allows for an easy decline in body temperature, which can also benefit the immune system, as an individual may be vulnerable to minor illnesses if the body temperature drops too abruptly after physical exercise.[151]
History
The benefits of exercise have been known since antiquity. Dating back to 65 BCE, it was Marcus Cicero, Roman politician and lawyer, who stated: "It is exercise alone that supports the spirits, and keeps the mind in vigor."[152] Exercise was also seen to be valued later in history during the Dark Ages as a means of survival by the barbaric tribes of Northern Europe.[153]
More recently, exercise was regarded as a beneficial force in the 19th century. After 1860, Archibald MacLaren opened a gymnasium at the University of Oxford and instituted a training regimen for 12 military officials at the university. This regimen was later assimilated into the training of the British Army.[154] Several mass exercise movements were started in the early twentieth century as well. The first and most significant of these in the UK was the Women's League of Health and Beauty, founded in 1930 by Mary Bagot Stack, that had 166,000 members in 1937.[155]
However, the link between physical health and exercise (or lack of it) was only discovered in 1949 and reported in 1953 by a team led by Jerry Morris.[156][157] Dr. Morris noted that men of similar social class and occupation (bus conductors versus bus drivers) had markedly different rates of heart attacks, depending on the level of exercise they got: bus drivers had a sedentary occupation and a higher incidence of heart disease, while bus conductors were forced to move continually and had a lower incidence of heart disease.[157] This link had not previously been noted and was later confirmed by other researchers.
Other animals
Physical exercise has been shown to benefit a wide range of other mammals, as well as salmon, juvenile crocodiles, and at least one species of bird.[158]
However, several studies have shown that lizards display no benefit from exercise, leading them to be termed "metabolically inflexible".[159] Indeed, damage from overtraining may occur following weeks of forced treadmill exercise in lizards.[159]
A number of studies of both rodents and humans have demonstrated that individual differences in both ability and propensity for exercise (i.e., voluntary exercise) have some genetic basis.[160][161]
Several studies of rodents have demonstrated that maternal[162] or juvenile access to wheels that allow voluntary exercise can increase the propensity to run as adults.[163] These studies further suggest that physical activity may be more "programmable" (for discussion, see Thrifty phenotype) than food intake.[164]
See also
Notes
- ^ Examples of these long-term effects include: marked improvements in executive function across all cognitive domains and small or moderate improvements in multiple forms of memory;[50][126][54] increased gray matter volume in the hippocampus, prefrontal cortex, components of the basal ganglia, and other structures;[50][54][127] and increased neural efficiency and greater functional connectivity between the left and right halves of the prefrontal cortex, the hippocampus and cingulate cortex.[50][128]
References
- ^ Kylasov A, Gavrov S (2011). Diversity Of Sport: non-destructive evaluation. Paris: UNESCO: Encyclopedia of Life Support Systems. pp. 462–491. ISBN 978-5-8931-7227-0.
- ^ Stampfer MJ, Hu FB, Manson JE, Rimm EB, Willett WC; Hu; Manson; Rimm; Willett (2000). "Primary Prevention of Coronary Heart Disease in Women through Diet and Lifestyle". New England Journal of Medicine. 343 (1): 16–22. doi:10.1056/NEJM200007063430103. PMID 10882764.
Conclusions[:] Among women, adherence to lifestyle guidelines involving diet, exercise, and abstinence from smoking is associated with a very low risk of coronary heart disease.
{{cite journal}}
: CS1 maint: multiple names: authors list (link) - ^ Hu FB, Manson JE, Stampfer MJ, Colditz G, Liu S, Solomon CG, Willett WC; Manson; Stampfer; Colditz; Liu; Solomon; Willett (2001). "Diet, lifestyle, and the risk of type 2 diabetes mellitus in women". The New England Journal of Medicine. 345 (11): 790–797. doi:10.1056/NEJMoa010492. PMID 11556298.
We followed 84,941 female nurses from 1980 to 1996 [...]. During 16 years of follow-up, we documented 3300 new cases of type 2 diabetes. Overweight or obesity was the single most important predictor of diabetes. Lack of exercise, a poor diet, current smoking, and abstinence from alcohol use were all associated with a significantly increased risk of diabetes, even after adjustment for the body-mass index.
{{cite journal}}
: CS1 maint: multiple names: authors list (link) - ^ "Physical Exercise". Health Appointment.
- ^ "Exercise". medical-dictionary.thefreedictionary.com. In turn citing: Gale Encyclopedia of Medicine. (2008): "Strengthening exercise increases muscle strength and mass, bone strength, and the body's metabolism. It can help attain and maintain proper weight and improve body image and self-esteem"
- ^ "Diet, Exercise, and Sleep". National Sleep Foundation.
- ^ Compare: "WHO: Obesity and overweight". who.int.
The Political Declaration of the High Level Meeting of the United Nations General Assembly on the Prevention and Control of Noncommunicable Diseases of September 2011, recognizes the critical importance of reducing unhealthy diet and physical inactivity.
- ^ Department of Physical Education, Sports Science and Recreation Management, Loughborough University, UK (2002). "The time course of the human growth hormone response to a 6 s and a 30 s cycle ergometer sprint". J Sports Sci. 20 (6): 487–94. doi:10.1080/02640410252925152. PMID 12137178.
{{cite journal}}
: CS1 maint: multiple names: authors list (link) - ^ Pimlott N (May 2010). "The miracle drug". Can Fam Physician. 56 (5): 407, 409. PMC 2868602. PMID 20463262.
- ^ a b c Bergstrom, Kristine; Muse, Toby; Tsai, Michelle; Strangio, Sebastian. "Fitness for Foreigners". Slate Magazine. Slate Magazine. Retrieved 5 December 2016.
- ^ a b Buckley, J. P.; Hedge, A.; Yates, T.; Copeland, R. J.; Loosemore, M.; Hamer, M.; Bradley, G.; Dunstan, D. W. (1 June 2015). "The sedentary office: a growing case for change towards better health and productivity. Expert statement commissioned by Public Health England and the Active Working Community Interest Company". British Journal of Sports Medicine. 49 (21): 1357–62. doi:10.1136/bjsports-2015-094618. PMID 26034192.
- ^ a b Compare: Meyers, Jonathan (2003). "Exercise and Cardiovascular Health". Circulation. 107: 2e–5. doi:10.1161/01.cir.0000048890.59383.8d. "The Surgeon General’s Report, a joint CDC/ACSM consensus statement, and a National Institutes of Health report agreed that the benefits mentioned above will generally occur by engaging in at least 30 minutes of modest activity on most, preferably all, days of the week. Modest activity is defined as any activity that is similar in intensity to brisk walking at a rate of about 3 to 4 miles per hour."
- ^ a b c d e f g h i National Institutes of Health, National Heart, Lung, and Blood Institute (June 2006). "Your Guide to Physical Activity and Your Heart" (PDF). U.S. Department of Health and Human Services.
{{cite web}}
: CS1 maint: multiple names: authors list (link) - ^ Wilmore J.; Knuttgen H. (2003). "Aerobic Exercise and Endurance Improving Fitness for Health Benefits". The Physician and Sportsmedicine. 31 (5): 45. doi:10.3810/psm.2003.05.367.
- ^ De Vos N.; Singh N.; Ross D.; Stavrinos T. (2005). "Optimal Load for Increasing Muscle Power During Explosive Resistance Training in Older Adults". The Journals of Gerontology. 60A (5): 638–647. doi:10.1093/gerona/60.5.638.
- ^ O'Connor D.; Crowe M.; Spinks W. (2005). "Effects of static stretching on leg capacity during cycling". Turin. 46 (1): 52–56.
- ^ "What Is Fitness?" (PDF). The CrossFit Journal. October 2002. p. 4. Retrieved 12 September 2010.
- ^ de Souza Nery S, Gomides RS, da Silva GV, de Moraes Forjaz CL, Mion D Jr, Tinucci T (1 March 2010). "Intra-Arterial Blood Pressure Response in Hypertensive Subjects during Low- and High-Intensity Resistance Exercise". Clinics. 65 (3): 271–7. doi:10.1590/S1807-59322010000300006. PMC 2845767. PMID 20360917.
{{cite journal}}
: CS1 maint: multiple names: authors list (link) - ^ a b c d e f g h i j k l m n o p q r s t u v w x y z aa Egan B, Zierath JR (February 2013). "Exercise metabolism and the molecular regulation of skeletal muscle adaptation". Cell Metabolism. 17 (2): 162–184. doi:10.1016/j.cmet.2012.12.012. PMID 23395166.
- ^ Gremeaux, V; Gayda, M; Lepers, R; Sosner, P; Juneau, M; Nigam, A (December 2012). "Exercise and longevity". Maturitas. 73 (4): 312–7. doi:10.1016/j.maturitas.2012.09.012. PMID 23063021.
- ^ Department Of Health And Human Services, United States (1996). "Physical Activity and Health". United States Department of Health. ISBN 9781428927940.
- ^ Woods, Jeffrey A.; Wilund, Kenneth R.; Martin, Stephen A.; Kistler, Brandon M. (29 October 2011). "Exercise, Inflammation and Aging". Aging and Disease. 3 (1): 130–140. PMC 3320801. PMID 22500274.
- ^ a b Kyu, Hmwe H; Bachman, Victoria F; Alexander, Lily T; Mumford, John Everett; Afshin, Ashkan; Estep, Kara; Veerman, J Lennert; Delwiche, Kristen; Iannarone, Marissa L; Moyer, Madeline L; Cercy, Kelly; Vos, Theo; Murray, Christopher J L; Forouzanfar, Mohammad H (9 August 2016). "Physical activity and risk of breast cancer, colon cancer, diabetes, ischemic heart disease, and ischemic stroke events: systematic review and dose-response meta-analysis for the Global Burden of Disease Study 2013". BMJ. 354: i3857. doi:10.1136/bmj.i3857. PMC 4979358. PMID 27510511.
- ^ a b Lee, I-Min; Shiroma, Eric J; Lobelo, Felipe; Puska, Pekka; Blair, Steven N; Katzmarzyk, Peter T (21 July 2012). "Impact of Physical Inactivity on the World's Major Non-Communicable Diseases". Lancet. 380 (9838): 219–229. doi:10.1016/S0140-6736(12)61031-9. PMC 3645500. PMID 22818936.
- ^ a b Dobbins, Maureen; Husson, Heather; DeCorby, Kara; LaRocca, Rebecca L (28 February 2013). Cochrane Database of Systematic Reviews. John Wiley & Sons, Ltd. doi:10.1002/14651858.cd007651.pub2.
- ^ Hubal MJ, Gordish-Dressman H, Thompson PD, Price TB, Hoffman EP, Angelopoulos TJ, Gordon PM, Moyna NM, Pescatello LS, Visich PS, Zoeller RF, Seip RL, Clarkson PM; Gordish-Dressman; Thompson; Price; Hoffman; Angelopoulos; Gordon; Moyna; Pescatello; Visich; Zoeller; Seip; Clarkson (June 2005). "Variability in muscle size and strength gain after unilateral resistance training". Medicine and Science in Sports and Exercise. 37 (6): 964–972. PMID 15947721.
{{cite journal}}
: CS1 maint: multiple names: authors list (link) - ^ Brutsaert TD, Parra EJ (2006). "What makes a champion? Explaining variation in human athletic performance". Respiratory Physiology & Neurobiology. 151 (2–3): 109–123. doi:10.1016/j.resp.2005.12.013. PMID 16448865.
- ^ Geddes, Linda (28 July 2007). "Superhuman". New Scientist. pp. 35–41.
- ^ "Being active combats risk of functional problems".
- ^ Physical Activity and Health. Diane Publishing. 1996.
- ^ Lumeng, Julie C (2006). "Small-group physical education classes result in important health benefits". The Journal of Pediatrics. 148 (3): 418–419. doi:10.1016/j.jpeds.2006.02.025.
- ^ Ahaneku, Joseph E.; Nwosu, Cosmas M.; Ahaneku, Gladys I. (2000). "Academic Stress and Cardiovascular Health". Academic Medicine. doi:10.1097/00001888-200006000-00002.
- ^ Fletcher, G. F; Balady, G; Blair, S. N; Blumenthal, J; Caspersen, C; Chaitman, B; Epstein, S; Froelicher, E. S. S; Froelicher, V. F; Pina, I. L; Pollock, M. L (1996). "Statement on Exercise: Benefits and Recommendations for Physical Activity Programs for All Americans: A Statement for Health Professionals by the Committee on Exercise and Cardiac Rehabilitation of the Council on Clinical Cardiology, American Heart Association". Circulation. 94 (4): 857. doi:10.1161/01.CIR.94.4.857. PMID 8772712.
- ^ Goodman, C. C.; Kapasi, Z. F. (2002). "The effect of exercise on the immune system". Rehabilitation Oncology.
- ^ a b c Gleeson M (August 2007). "Immune function in sport and exercise". J. Appl. Physiol. 103 (2): 693–9. doi:10.1152/japplphysiol.00008.2007. PMID 17303714.
- ^ Swardfager W (2012). "Exercise intervention and inflammatory markers in coronary artery disease: a meta-analysis". Am. Heart J. 163 (4): 666–76. doi:10.1016/j.ahj.2011.12.017. PMID 22520533.
- ^ Ballard-Barbash R, Friedenreich CM, Courneya KS, Siddiqi SM, McTiernan A, Alfano CM (2012). "Physical Activity, Biomarkers, and Disease Outcomes in Cancer Survivors: A Systematic Review". JNCI Journal of the National Cancer Institute. 104 (11): 815–840. doi:10.1093/jnci/djs207. PMC 3465697. PMID 22570317.
- ^ a b Yuasa Y, Nagasaki H, Akiyama Y, Hashimoto Y, Takizawa T, Kojima K, et al. (2009). "DNA methylation status is inversely correlated with green tea intake and physical activity in gastric cancer patients". Int. J. Cancer. 124 (11): 2677–82. doi:10.1002/ijc.24231. PMID 19170207.
- ^ a b Zeng H, Irwin ML, Lu L, Risch H, Mayne S, Mu L, Deng Q, Scarampi L, Mitidieri M, Katsaros D, Yu H (May 2012). "Physical activity and breast cancer survival: an epigenetic link through reduced methylation of a tumor suppressor gene L3MBTL1". Breast Cancer Res Treat. 133 (1): 127–35. doi:10.1007/s10549-011-1716-7. PMID 21837478.
- ^ Li J, Bench AJ, Piltz S, Vassiliou G, Baxter EJ, Ferguson-Smith AC, Green AR (October 2005). "L3mbtl, the mouse orthologue of the imprinted L3MBTL, displays a complex pattern of alternative splicing and escapes genomic imprinting". Genomics. 86 (4): 489–94. doi:10.1016/j.ygeno.2005.06.012. PMID 16081246.
- ^ Bench AJ, Li J, Huntly BJ, Delabesse E, Fourouclas N, Hunt AR, Deloukas P, Green AR (December 2004). "Characterization of the imprinted polycomb gene L3MBTL, a candidate 20q tumour suppressor gene, in patients with myeloid malignancies". Br J Haematol. 127 (5): 509–18. doi:10.1111/j.1365-2141.2004.05278.x. PMID 15566354.
- ^ Bryan AD, Magnan RE, Hooper AE, Harlaar N, Hutchison KE (2013). "Physical activity and differential methylation of breast cancer genes assayed from saliva: a preliminary investigation". Ann Behav Med. 45 (1): 89–98. doi:10.1007/s12160-012-9411-4. PMC 3548059. PMID 23054940.
- ^ Lira FS, Neto JC, Seelaender M (June 2014). "Exercise training as treatment in cancer cachexia". Appl Physiol Nutr Metab. 39 (6): 679–86. doi:10.1139/apnm-2013-0554. PMID 24797380.
- ^ Evans WJ, Morley JE, Argiles J, Bales C, Baracos V, Guttridge D, et al. (2008). "Cachexia: a new definition". Clin Nutr. 27 (6): 793–799. doi:10.1016/j.clnu.2008.06.013. PMID 18718696.
- ^ Sandri M, et al. (2006). "PGC-1α protects skeletal muscle from atrophy by suppressing FoxO3 action and atrophy-specific gene transcription". Proc. Natl. Acad. Sci. USA. 103 (44): 16260–16265. Bibcode:2006PNAS..10316260S. doi:10.1073/pnas.0607795103. PMC 1637570. PMID 17053067.
- ^ Brault J. J.; Jespersen J. G.; Goldberg A. L. (2010). "Peroxisome proliferator-activated receptor γ coactivator 1α or 1β overexpression inhibits muscle protein degradation, induction of ubiquitin ligases, and disuse atrophy". J. Biol. Chem. 285 (25): 19460–19471. doi:10.1074/jbc.m110.113092. PMC 2885225. PMID 20404331.
{{cite journal}}
: CS1 maint: unflagged free DOI (link) - ^ Ruas J. L.; et al. (2012). "A PGC-1α isoform induced by resistance training regulates skeletal muscle hypertrophy". Cell. 151 (6): 1319–1331. doi:10.1016/j.cell.2012.10.050.
- ^ Vissing K, et al. (2013). "Effect of resistance exercise contraction mode and protein supplementation on members of the STARS signalling pathway". J. Physiol. 591 (15): 3749–3763. doi:10.1113/jphysiol.2012.249755.
- ^ Ferrara N, et al. (2008). "Exercise training promotes SIRT1 activity in aged rats". Rejuven. Res. 11: 139–150. doi:10.1089/rej.2007.0576.
- ^ a b c d e Erickson KI, Hillman CH, Kramer AF (August 2015). "Physical activity, brain, and cognition". Current Opinion in Behavioral Sciences. 4: 27–32. doi:10.1016/j.cobeha.2015.01.005. S2CID 54301951.
- ^ Paillard T, Rolland Y, de Souto Barreto P (July 2015). "Protective Effects of Physical Exercise in Alzheimer's Disease and Parkinson's Disease: A Narrative Review". J Clin Neurol. 11 (3): 212–219. doi:10.3988/jcn.2015.11.3.212. PMC 4507374. PMID 26174783.
- ^ McKee AC, Daneshvar DH, Alvarez VE, Stein TD (January 2014). "The neuropathology of sport". Acta Neuropathol. 127 (1): 29–51. doi:10.1007/s00401-013-1230-6. PMC 4255282. PMID 24366527.
- ^ Denham J, Marques FZ, O'Brien BJ, Charchar FJ (February 2014). "Exercise: putting action into our epigenome". Sports Med. 44 (2): 189–209. doi:10.1007/s40279-013-0114-1. PMID 24163284. S2CID 30210091.
- ^ a b c d e Gomez-Pinilla F, Hillman C (January 2013). "The influence of exercise on cognitive abilities". Comprehensive Physiology. 3 (1): 403–428. doi:10.1002/cphy.c110063. ISBN 9780470650714. PMC 3951958. PMID 23720292.
- ^ Buckley J, Cohen JD, Kramer AF, McAuley E, Mullen SP (2014). "Cognitive control in the self-regulation of physical activity and sedentary behavior". Front Hum Neurosci. 8: 747. doi:10.3389/fnhum.2014.00747. PMC 4179677. PMID 25324754.
- ^ a b c Cox EP, O'Dwyer N, Cook R, Vetter M, Cheng HL, Rooney K, O'Connor H (August 2016). "Relationship between physical activity and cognitive function in apparently healthy young to middle-aged adults: A systematic review". J. Sci. Med. Sport. 19 (8): 616–628. doi:10.1016/j.jsams.2015.09.003. PMID 26552574.
- ^ CDC (1 August 2023). "Benefits of Physical Activity". Centers for Disease Control and Prevention. Retrieved 7 December 2023.
- ^ a b c d Schuch FB, Vancampfort D, Rosenbaum S, Richards J, Ward PB, Stubbs B (July 2016). "Exercise improves physical and psychological quality of life in people with depression: A meta-analysis including the evaluation of control group response". Psychiatry Res. 241: 47–54. doi:10.1016/j.psychres.2016.04.054. PMID 27155287. S2CID 4787287.
- ^ Pratali L, Mastorci F, Vitiello N, Sironi A, Gastaldelli A, Gemignani A (November 2014). "Motor Activity in Aging: An Integrated Approach for Better Quality of Life". International Scholarly Research Notices. 2014: 257248. doi:10.1155/2014/257248. PMC 4897547. PMID 27351018.
- ^ Mandolesi L, Polverino A, Montuori S, Foti F, Ferraioli G, Sorrentino P, Sorrentino G (27 April 2018). "Effects of Physical Exercise on Cognitive Functioning and Wellbeing: Biological and Psychological Benefits". Frontiers in Psychology. 9: 509. doi:10.3389/fpsyg.2018.00509. PMC 5934999. PMID 29755380.
- ^ a b c Basso JC, Suzuki WA (March 2017). "The Effects of Acute Exercise on Mood, Cognition, Neurophysiology, and Neurochemical Pathways: A Review". Brain Plasticity. 2 (2): 127–152. doi:10.3233/BPL-160040. PMC 5928534. PMID 29765853.
- ^ "Exercise and mental health". betterhealth.vic.gov.au. Department of Health & Human Services. Retrieved 19 November 2022.
- ^ "Exercise and Mental Health". Exercise Psychology: 93–94. 2013. doi:10.5040/9781492595502.part-002. ISBN 9781492595502.
- ^ "10 great reasons to love aerobic exercise". Mayo Clinic. Retrieved 5 December 2023.
- ^ a b Josefsson T, Lindwall M, Archer T (2014). "Physical exercise intervention in depressive disorders: meta-analysis and systematic review". Scand J Med Sci Sports. 24 (2): 259–272. doi:10.1111/sms.12050. PMID 23362828. S2CID 29351791.
- ^ a b c Mura G, Moro MF, Patten SB, Carta MG (2014). "Exercise as an add-on strategy for the treatment of major depressive disorder: a systematic review". CNS Spectr. 19 (6): 496–508. doi:10.1017/S1092852913000953. PMID 24589012. S2CID 32304140.
- ^ Den Heijer AE, Groen Y, Tucha L, Fuermaier AB, Koerts J, Lange KW, Thome J, Tucha O (July 2016). "Sweat it out? The effects of physical exercise on cognition and behavior in children and adults with ADHD: a systematic literature review". J. Neural Transm. (Vienna). 124 (Suppl 1): 3–26. doi:10.1007/s00702-016-1593-7. PMC 5281644. PMID 27400928.
- ^ Petersen RC, Lopez O, Armstrong MJ, Getchius T, Ganguli M, Gloss D, Gronseth GS, Marson D, Pringsheim T, Day GS, Sager M, Stevens J, Rae-Grant A (January 2018). "Practice guideline update summary: Mild cognitive impairment – Report of the Guideline Development, Dissemination, and Implementation Subcommittee of the American Academy of Neurology". Neurology. Special article. 90 (3): 126–135. doi:10.1212/WNL.0000000000004826. PMC 5772157. PMID 29282327.
- ^ Carroll ME, Smethells JR (February 2016). "Sex Differences in Behavioral Dyscontrol: Role in Drug Addiction and Novel Treatments". Front. Psychiatry. 6: 175. doi:10.3389/fpsyt.2015.00175. PMC 4745113. PMID 26903885.
- ^ Lynch WJ, Peterson AB, Sanchez V, Abel J, Smith MA (September 2013). "Exercise as a novel treatment for drug addiction: a neurobiological and stage-dependent hypothesis". Neurosci Biobehav Rev. 37 (8): 1622–1644. doi:10.1016/j.neubiorev.2013.06.011. PMC 3788047. PMID 23806439.
- ^ Olsen CM (December 2011). "Natural rewards, neuroplasticity, and non-drug addictions". Neuropharmacology. 61 (7): 1109–1122. doi:10.1016/j.neuropharm.2011.03.010. PMC 3139704. PMID 21459101.
- ^ Linke SE, Ussher M (2015). "Exercise-based treatments for substance use disorders: evidence, theory, and practicality". Am J Drug Alcohol Abuse. 41 (1): 7–15. doi:10.3109/00952990.2014.976708. PMC 4831948. PMID 25397661.
- ^ Farina N, Rusted J, Tabet N (January 2014). "The effect of exercise interventions on cognitive outcome in Alzheimer's disease: a systematic review". Int Psychogeriatr. 26 (1): 9–18. doi:10.1017/S1041610213001385. PMID 23962667. S2CID 24936334.
- ^ Tomlinson CL, Patel S, Meek C, Herd CP, Clarke CE, Stowe R, Shah L, Sackley CM, Deane KH, Wheatley K, Ives N (September 2013). "Physiotherapy versus placebo or no intervention in Parkinson's disease". Cochrane Database Syst Rev. 9 (9): CD002817. doi:10.1002/14651858.CD002817.pub4. PMC 7120224. PMID 24018704.
- ^ Blondell SJ, Hammersley-Mather R, Veerman JL (May 2014). "Does physical activity prevent cognitive decline and dementia?: A systematic review and meta-analysis of longitudinal studies". BMC Public Health. 14: 510. doi:10.1186/1471-2458-14-510. PMC 4064273. PMID 24885250.
- ^ Cite error: The named reference
Physical activity intervention
was invoked but never defined (see the help page). - ^ Cite error: The named reference
PA-dep June 2015
was invoked but never defined (see the help page). - ^ Gong H, Ni C, Shen X, Wu T, Jiang C (February 2015). "Yoga for prenatal depression: a systematic review and meta-analysis". BMC Psychiatry. 15: 14. doi:10.1186/s12888-015-0393-1. PMC 4323231. PMID 25652267.
{{cite journal}}
: CS1 maint: unflagged free DOI (link) - ^ Cite error: The named reference
BDNF depression
was invoked but never defined (see the help page). - ^ Tantimonaco M, Ceci R, Sabatini S, Catani MV, Rossi A, Gasperi V, Maccarrone M (2014). "Physical activity and the endocannabinoid system: an overview". Cell. Mol. Life Sci. 71 (14): 2681–2698. doi:10.1007/s00018-014-1575-6. PMID 24526057.
- ^ Dinas PC, Koutedakis Y, Flouris AD (2011). "Effects of exercise and physical activity on depression". Ir J Med Sci. 180 (2): 319–325. doi:10.1007/s11845-010-0633-9. PMID 21076975.
- ^ Szabo A, Billett E, Turner J (2001). "Phenylethylamine, a possible link to the antidepressant effects of exercise?". Br J Sports Med. 35 (5): 342–343. doi:10.1136/bjsm.35.5.342. PMC 1724404. PMID 11579070.
- ^ Lindemann L, Hoener MC (2005). "A renaissance in trace amines inspired by a novel GPCR family". Trends Pharmacol. Sci. 26 (5): 274–281. doi:10.1016/j.tips.2005.03.007. PMID 15860375.
- ^ Berry MD (2007). "The potential of trace amines and their receptors for treating neurological and psychiatric diseases". Rev Recent Clin Trials. 2 (1): 3–19. doi:10.2174/157488707779318107. PMID 18473983.
- ^ a b Teychenne M, Costigan SA, Parker K (June 2015). "The association between sedentary behaviour and risk of anxiety: a systematic review". BMC Public Health. 15: 513. doi:10.1186/s12889-015-1843-x. PMC 4474345. PMID 26088005.
{{cite journal}}
: CS1 maint: unflagged free DOI (link) - ^ Buman, M.P., King, A.C. (2010). "Exercise as a Treatment to Enhance Sleep". American Journal of Lifestyle Medicine. 31 (5): 514. doi:10.1177/1559827610375532.
{{cite journal}}
: CS1 maint: multiple names: authors list (link) - ^ Youngstedt, S.D. (April 2005). "Effects of exercise on sleep" (PDF). Clin Sports Med. 24 (2): 355–65, xi. doi:10.1016/j.csm.2004.12.003. Retrieved 9 April 2012.
- ^ Alexander, C. 1998. Cutting weight, losing life. News & Observer, February 8: 1998, A.1. Retrieved 5 October 2006, from ProQuest database.
- ^ Möhlenkamp S, Lehmann N, Breuckmann F, Bröcker-Preuss M, Nassenstein K, Halle M, Budde T, Mann K, Barkhausen J, Heusch G, Jöckel KH, Erbel R (200). "Running: the risk of coronary events : Prevalence and prognostic relevance of coronary atherosclerosis in marathon runners". Eur. Heart J. 29 (15): 1903–10. doi:10.1093/eurheartj/ehn163. PMID 18426850.
- ^ Benito B, Gay-Jordi G, Serrano-Mollar A, Guasch E, Shi Y, Tardif JC, Brugada J, Nattel S, Mont L (2011). "Cardiac arrhythmogenic remodeling in a rat model of long-term intensive exercise training". Circulation. 123 (1): 13–22. doi:10.1161/CIRCULATIONAHA.110.938282. PMID 21173356.
- ^ Wilson M, O'Hanlon R, Prasad S, Deighan A, Macmillan P, Oxborough D, Godfrey R, Smith G, Maceira A, Sharma S, George K, Whyte G (2011). "Diverse patterns of myocardial fibrosis in lifelong, veteran endurance athletes". J Appl Physiol. 110 (6): 1622–6. doi:10.1152/japplphysiol.01280.2010. PMC 3119133. PMID 21330616.
- ^ O'Keefe JH, Patil HR, Lavie CJ, Magalski A, Vogel RA, McCullough PA (2012). "Potential Adverse Cardiovascular Effects from Excessive Endurance Exercise". Mayo Clinic Proceedings. 87 (6): 587–595. doi:10.1016/j.mayocp.2012.04.005. PMC 3538475. PMID 22677079.
- ^ Aertsens J, de Geus B, Vandenbulcke G, Degraeuwe B, Broekx S, De Nocker L, Liekens I, Mayeres I, Meeusen R, Thomas I, Torfs R, Willems H, Int Panis L (2010). "Commuting by bike in Belgium, the costs of minor accidents". Accident Analysis and Prevention. 42 (6): 2149–2157. doi:10.1016/j.aap.2010.07.008. PMID 20728675.
- ^ Int Panis, L; De Geus, Bas; Vandenbulcke, GréGory; Willems, Hanny; Degraeuwe, Bart; Bleux, Nico; Mishra, Vinit; Thomas, Isabelle; Meeusen, Romain (2010). "Exposure to particulate matter in traffic: A comparison of cyclists and car passengers". Atmospheric Environment. 44 (19): 2263–2270. Bibcode:2010AtmEn..44.2263I. doi:10.1016/j.atmosenv.2010.04.028.
- ^ Jacobs L, Nawrot TS, de Geus B, Meeusen R, Degraeuwe B, Bernard A, Sughis M, Nemery B, Panis LI (October 2010). "Subclinical responses in healthy cyclists briefly exposed to traffic-related air pollution". Environmental Health. 9 (64): 64. doi:10.1186/1476-069X-9-64. PMC 2984475. PMID 20973949.
{{cite journal}}
: CS1 maint: unflagged free DOI (link) - ^ Jimenez C.; Pacheco E.; Moreno A.; Carpenter A. (1996). "A Soldier's Neck and Shoulder Pain". The Physician and Sportsmedicine. 24 (6): 81–82. doi:10.3810/psm.1996.06.1384.
- ^ Smith L.L. (2003). "Overtraining, excessive exercise, and altered immunity, 2003". Sports Medicine. 33 (5): 347–364. doi:10.2165/00007256-200333050-00002.
- ^ Fredericson M and Misra AK. (2007) Epidemiology and aetiology of marathon running injuries. Sports Med. 37(4-5). 437–439
- ^ Furia, John. "The Female Athlete Triad". Medscape.com.
- ^ http://www.listfitness.com/exercise-excessively-addiction/
- ^ Koivula, N, Hassmén, P and Fallby, J. (2002) Self-esteem and perfectionism in elite athletes: effects on competitive anxiety and self-confidence. Personality and Individual Differences.
- ^ a b c d e f g h Brook MS, Wilkinson DJ, Phillips BE, Perez-Schindler J, Philp A, Smith K, Atherton PJ (January 2016). "Skeletal muscle homeostasis and plasticity in youth and ageing: impact of nutrition and exercise". Acta Physiologica. 216 (1): 15–41. doi:10.1111/apha.12532. PMC 4843955. PMID 26010896.
- ^ a b c Phillips SM (May 2014). "A brief review of critical processes in exercise-induced muscular hypertrophy". Sports Med. 44 Suppl 1: S71–S77. doi:10.1007/s40279-014-0152-3. PMC 4008813. PMID 24791918.
- ^ Brioche T, Pagano AF, Py G, Chopard A (April 2016). "Muscle wasting and aging: Experimental models, fatty infiltrations, and prevention". Molecular Aspects of Medicine. 50: 56–87. doi:10.1016/j.mam.2016.04.006. PMID 27106402.
- ^ a b Wilkinson DJ, Hossain T, Hill DS, Phillips BE, Crossland H, Williams J, Loughna P, Churchward-Venne TA, Breen L, Phillips SM, Etheridge T, Rathmacher JA, Smith K, Szewczyk NJ, Atherton PJ (June 2013). "Effects of leucine and its metabolite β-hydroxy-β-methylbutyrate on human skeletal muscle protein metabolism" (PDF). J. Physiol. 591 (11): 2911–2923. doi:10.1113/jphysiol.2013.253203. PMC 3690694. PMID 23551944. Retrieved 27 May 2016.
- ^ Phillips SM (July 2015). "Nutritional supplements in support of resistance exercise to counter age-related sarcopenia". Adv. Nutr. 6 (4): 452–460. doi:10.3945/an.115.008367. PMC 4496741. PMID 26178029.
- ^ a b Boushel R, Lundby C, Qvortrup K, Sahlin K (October 2014). "Mitochondrial plasticity with exercise training and extreme environments". Exerc. Sport Sci. Rev. 42 (4): 169–174. doi:10.1249/JES.0000000000000025. PMID 25062000.
- ^ Valero T (2014). "Mitochondrial biogenesis: pharmacological approaches". Curr. Pharm. Des. 20 (35): 5507–5509. doi:10.2174/138161282035140911142118. PMID 24606795.
- ^ Lipton JO, Sahin M (October 2014). "The neurology of mTOR". Neuron. 84 (2): 275–291. doi:10.1016/j.neuron.2014.09.034. PMC 4223653. PMID 25374355.
Figure 2: The mTOR Signaling Pathway - ^ a b Wang, E; Næss, MS; Hoff, J; Albert, TL; Pham, Q; Richardson, RS; Helgerud, J (16 November 2013). "Exercise-training-induced changes in metabolic capacity with age: the role of central cardiovascular plasticity". Age (Dordrecht, Netherlands). 36 (2): 665–676. doi:10.1007/s11357-013-9596-x. PMC 4039249. PMID 24243396.
- ^ Potempa, K; Lopez, M; Braun, LT; Szidon, JP; Fogg, L; Tincknell, T (January 1995). "Physiological outcomes of aerobic exercise training in hemiparetic stroke patients". Stroke: A Journal of Cerebral Circulation. 26 (1): 101–5. doi:10.1161/01.str.26.1.101. PMID 7839377.
- ^ Wilmore, JH; Stanforth, PR; Gagnon, J; Leon, AS; Rao, DC; Skinner, JS; Bouchard, C (July 1996). "Endurance exercise training has a minimal effect on resting heart rate: the HERITAGE Study". Medicine and science in sports and exercise. 28 (7): 829–35. doi:10.1097/00005768-199607000-00009. PMID 8832536.
- ^ Carter, JB; Banister, EW; Blaber, AP (2003). "Effect of endurance exercise on autonomic control of heart rate". Sports Medicine. 33 (1): 33–46. doi:10.2165/00007256-200333010-00003. PMID 12477376.
- ^ Chen, Chao‐Yin; Dicarlo, Stephen E. (January 1998). "Endurance exercise training‐induced resting Bradycardia: A brief review". Sports Medicine, Training and Rehabilitation. 8 (1): 37–77. doi:10.1080/15438629709512518.
- ^ Crewther, BT; Heke, TL; Keogh, JW (February 2013). "The effects of a resistance-training program on strength, body composition and baseline hormones in male athletes training concurrently for rugby union 7's". The Journal of sports medicine and physical fitness. 53 (1): 34–41. PMID 23470909.
- ^ Schoenfeld, BJ (June 2013). "Postexercise hypertrophic adaptations: a reexamination of the hormone hypothesis and its applicability to resistance training program design". Journal of strength and conditioning research / National Strength & Conditioning Association. 27 (6): 1720–30. doi:10.1519/JSC.0b013e31828ddd53. PMID 23442269.
- ^ Dalgas, U; Stenager, E; Lund, C; Rasmussen, C; Petersen, T; Sørensen, H; Ingemann-Hansen, T; Overgaard, K (July 2013). "Neural drive increases following resistance training in patients with multiple sclerosis". Journal of Neurology. 260 (7): 1822–32. doi:10.1007/s00415-013-6884-4. PMID 23483214.
- ^ Staron, RS; Karapondo, DL; Kraemer, WJ; Fry, AC; Gordon, SE; Falkel, JE; Hagerman, FC; Hikida, RS (March 1994). "Skeletal muscle adaptations during early phase of heavy-resistance training in men and women". Journal of Applied Physiology. 76 (3): 1247–55. PMID 8005869.
- ^ Folland, JP; Williams, AG (2007). "The adaptations to strength training : morphological and neurological contributions to increased strength". Sports Medicine. 37 (2): 145–68. doi:10.2165/00007256-200737020-00004. PMID 17241104.
- ^ Moritani, T; deVries, HA (June 1979). "Neural factors versus hypertrophy in the time course of muscle strength gain". American journal of physical medicine. 58 (3): 115–30. PMID 453338.
- ^ Narici, MV; Roi, GS; Landoni, L; Minetti, AE; Cerretelli, P (1989). "Changes in force, cross-sectional area and neural activation during strength training and detraining of the human quadriceps". European journal of applied physiology and occupational physiology. 59 (4): 310–9. doi:10.1007/bf02388334. PMID 2583179.
- ^ Pedersen, BK (July 2013). "Muscle as a secretory organ". Comprehensive Physiology. 3 (3): 1337–62. doi:10.1002/cphy.c120033. ISBN 9780470650714. PMID 23897689.
- ^ Cohen S, Williamson GM; Williamson (1991). "Stress and infectious disease in humans". Psychological Bulletin. 109 (1): 5–24. doi:10.1037/0033-2909.109.1.5. PMID 2006229.
- ^ Borer KT, Wuorinen EC, Lukos JR, Denver JW, Porges SW, Burant CF; Wuorinen; Lukos; Denver; Porges; Burant (August 2009). "Two bouts of exercise before meals but not after meals, lower fasting blood glucose". Medicine in Science and Sports and Exercise. 41 (8): 1606–14. doi:10.1249/MSS.0b013e31819dfe14. PMID 19568199.
{{cite journal}}
: CS1 maint: multiple names: authors list (link) - ^ Wisløff U, Ellingsen Ø, Kemi OJ; Ellingsen; Kemi (July 2009). "High=Intensity Interval Training to Maximize Cardiac Benefit of Exercise Taining?". Exercise and Sports Sciences Reviews. 37 (3): 139–146. doi:10.1097/JES.0b013e3181aa65fc. PMID 19550205.
{{cite journal}}
: CS1 maint: multiple names: authors list (link) - ^ a b c Cite error: The named reference
Exercise - neurotrophic factors + basal ganglia
was invoked but never defined (see the help page). - ^ Cite error: The named reference
gray matter
was invoked but never defined (see the help page). - ^ Cite error: The named reference
exercise benefits
was invoked but never defined (see the help page). - ^ Cite error: The named reference
BDNF meta analysis
was invoked but never defined (see the help page). - ^ a b Bouchard J, Villeda SA (2015). "Aging and brain rejuvenation as systemic events". J. Neurochem. 132 (1): 5–19. doi:10.1111/jnc.12969. PMC 4301186. PMID 25327899.
- ^ Silverman MN, Deuster PA (October 2014). "Biological mechanisms underlying the role of physical fitness in health and resilience". Interface Focus. 4 (5): 20140040. doi:10.1098/rsfs.2014.0040. PMC 4142018. PMID 25285199.
- ^ Tarumi T, Zhang R (January 2014). "Cerebral hemodynamics of the aging brain: risk of Alzheimer disease and benefit of aerobic exercise". Front Physiol. 5: 6. doi:10.3389/fphys.2014.00006. PMC 3896879. PMID 24478719.
{{cite journal}}
: CS1 maint: unflagged free DOI (link) - ^ Aberg D (2010). "Role of the growth hormone/insulin-like growth factor 1 axis in neurogenesis". Endocr Dev. Endocrine Development. 17: 63–76. doi:10.1159/000262529. ISBN 978-3-8055-9302-1. PMID 19955757.
- ^ a b Baker, Philip R. A.; Francis, Daniel P.; Soares, Jesus; Weightman, Alison L.; Foster, Charles (1 January 2015). "Community wide interventions for increasing physical activity". The Cochrane Database of Systematic Reviews. 1: CD008366. doi:10.1002/14651858.CD008366.pub3. PMID 25556970.
- ^ Howe, Tracey E; Rochester, Lynn; Neil, Fiona; Skelton, Dawn A; Ballinger, Claire (9 November 2011). Cochrane Database of Systematic Reviews. John Wiley & Sons, Ltd. doi:10.1002/14651858.cd004963.pub3.
- ^ Liu, Chiung-ju; Latham, Nancy K (8 July 2009). Cochrane Database of Systematic Reviews. John Wiley & Sons, Ltd. doi:10.1002/14651858.cd002759.pub2/abstract.
- ^ Gc, V; Wilson, EC; Suhrcke, M; Hardeman, W; Sutton, S; VBI Programme, Team (April 2016). "Are brief interventions to increase physical activity cost-effective? A systematic review". British journal of sports medicine. 50 (7): 408–17. doi:10.1136/bjsports-2015-094655. PMC 4819643. PMID 26438429.
- ^ "The effectiveness of interventions to increase physical activity. A systematic review". Am J Prev Med. 22 (4 Suppl): 73–107. May 2002. doi:10.1016/S0749-3797(02)00434-8. PMID 11985936.
{{cite journal}}
: Unknown parameter|authors=
ignored (help) - ^ Durán, Víctor Hugo. "Stopping the rising tide of chronic diseases Everyone's Epidemic". Pan American Health Organization. paho.org. Retrieved 10 January 2009.
- ^ Baker, Philip RA; Dobbins, Maureen; Soares, Jesus; Francis, Daniel P; Weightman, Alison L; Costello, Joseph T (6 January 2015). Public health interventions for increasing physical activity in children, adolescents and adults: an overview of systematic reviews. John Wiley & Sons, Ltd. doi:10.1002/14651858.cd011454.
- ^ Reed, Jennifer L; Prince, Stephanie A; Cole, Christie A; Fodor, J; Hiremath, Swapnil; Mullen, Kerri-Anne; Tulloch, Heather E; Wright, Erica; Reid, Robert D (19 December 2014). "Workplace physical activity interventions and moderate-to-vigorous intensity physical activity levels among working-age women: a systematic review protocol". Systematic Reviews. 3 (1): 147. doi:10.1186/2046-4053-3-147. PMC 4290810. PMID 25526769.
{{cite journal}}
: CS1 maint: unflagged free DOI (link) - ^ Xu, Huilan; Wen, Li Ming; Rissel, Chris (19 March 2015). "Associations of Parental Influences with Physical Activity and Screen Time among Young Children: A Systematic Review". Journal of Obesity. 2015: 1–23. doi:10.1155/2015/546925. PMC 4383435. PMID 25874123.
{{cite journal}}
: CS1 maint: unflagged free DOI (link) - ^ "Youth Physical Activity Guidelines". Centers for Disease Control and Prevention.
- ^ a b "WHO: Obesity and overweight". World Health Organization. Archived from the original on 18 December 2008. Retrieved 10 January 2009.
- ^ Kennedy AB, Resnick PB (May 2015). "Mindfulness and Physical Activity". American Journal of Lifestyle Medicine. 9 (3): 3221–223. doi:10.1177/1559827614564546.
- ^ Hernandez, Javier (24 June 2008). "Car-Free Streets, a Colombian Export, Inspire Debate". NY Times. NY Times.
- ^ Sullivan, Nicky. "Gyms". Travel Fish. Travel Fish. Retrieved 8 December 2016.
- ^ Tatlow, Anita. "When in Sweden...making the most of the great outdoors!". Stockholm on a Shoestring. Stockholm on a Shoestring. Retrieved 5 December 2016.
- ^ Langfitt, Frank. "Beijing's Other Games: Dancing In The Park". National Public Radio. National Public Radio. Retrieved 5 December 2016.
- ^ Kimber N.; Heigenhauser G.; Spriet L.; Dyck D. (2003). "Skeletal muscle fat and carbohydrate metabolism during recovery from glycogen-depleting exercise in humans". American Journal of Lifestyle Medicine. 548 (3): 919–927. doi:10.1113/jphysiol.2002.031179.
- ^ Reilly T, Ekblom B (June 2005). "The use of recovery methods post-exercise". J. Sports Sci. 23 (6): 619–627. doi:10.1080/02640410400021302. PMID 16195010.
- ^ "Quotes About Exercise Top 10 List".
- ^ "History of Fitness". www.unm.edu. Retrieved 20 September 2017.
- ^ "physical culture". Encyclopedia Britannica. Retrieved 20 September 2017.
- ^ "The Fitness League History". The Fitness League. Archived from the original on 29 July 2009. Retrieved 8 April 2015.
{{cite web}}
: Unknown parameter|deadurl=
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suggested) (help) - ^ Kuper, Simon (11 September 2009). "The man who invented exercise". Financial Times. Retrieved 12 September 2009.
- ^ a b Morris JN, Heady JA, Raffle PA, Roberts CG, Parks JW (1953). "Coronary heart-disease and physical activity of work". Lancet. 265 (6795): 1053–7. doi:10.1016/S0140-6736(53)90665-5. PMID 13110049.
- ^ Owerkowicz T, Baudinette RV (2008). "Exercise training enhances aerobic capacity in juvenile estuarine crocodiles (Crocodylus porosus)". Comparative Biochemistry and Physiology A. 150 (2): 211–6. doi:10.1016/j.cbpa.2008.04.594. PMID 18504156.
- ^ a b Garland T, Else PL, Hulbert AJ, Tap P (1987). "Effects of endurance training and captivity on activity metabolism of lizards". Am. J. Physiol. 252 (3 Pt 2): R450–6. PMID 3826409.
{{cite journal}}
: CS1 maint: multiple names: authors list (link) - ^ Garland T, Schutz H, Chappell MA, Keeney BK, Meek TH, Copes LE, Acosta W, Drenowatz C, Maciel RC, van Dijk G, Kotz CM, Eisenmann JC (2011). "The biological control of voluntary exercise, spontaneous physical activity and daily energy expenditure in relation to obesity: human and rodent perspectives". J. Exp. Biol. 214 (Pt 2): 206–29. doi:10.1242/jeb.048397. PMC 3008631. PMID 21177942.
{{cite journal}}
: CS1 maint: multiple names: authors list (link) - ^ Kelly SA, Pomp D (June 2013). "Genetic determinants of voluntary exercise". Trends Genet. 29 (6): 348–57. doi:10.1016/j.tig.2012.12.007. PMC 3665695. PMID 23351966.
- ^ Eclarinal, J. D; Zhu, S; Baker, M. S; Piyarathna, D. B; Coarfa, C; Fiorotto, M. L; Waterland, R. A (2016). "Maternal exercise during pregnancy promotes physical activity in adult offspring". The FASEB Journal. 30 (7): 2541–2548. doi:10.1096/fj.201500018R. PMC 4904289.
{{cite journal}}
: CS1 maint: unflagged free DOI (link) - ^ Acosta, W.; Meek, T. H.; Schutz, H.; Dlugosz, E. M.; Vu, K. T.; Garland Jr, T. (2015). "Effects of early-onset voluntary exercise on adult physical activity and associated phenotypes in mice". Physiology & Behavior. 149: 279–286. doi:10.1016/j.physbeh.2015.06.020. PMID 26079567.
- ^ Zhu, S.; Eclarinal, J.; Baker, M. S.; Li, G.; Waterland, R. A. (2016). "Developmental programming of energy balance regulation: is physical activity more "programmable" than food intake?". Proceedings of the Nutrition Society. 75 (1): 73–77. doi:10.1017/s0029665115004127. PMID 26511431.
Further reading
- Bruno Pauletto (1991). Strength Training for Coaches. Champaign, IL: Leisure Press. p. 179. ISBN 0880113715. Retrieved 11 August 2017.
- Donatelle, Rebecca J. (2005). Health, The Basics (6th ed.). San Francisco: Pearson Education. ISBN 0-8053-2852-1.
- Hardman A, Stensel D (2009). Physical Activity and Health: The Evidence Explained. London: Routledge. ISBN 978-0-415-42198-0.
- Ainsworth BE, Haskell WL, Leon AS, Jacobs DR, Montoye HJ, Sallis JF, Paffenbarger RS; Haskell; Leon; Jacobs Jr; Montoye; Sallis; Paffenbarger Jr (1993). "Compendium of physical activities: Classification of energy costs of human physical activities". Medicine and Science in Sports and Exercise. 25 (1): 71–80. doi:10.1249/00005768-199301000-00011. PMID 8292105.
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: CS1 maint: multiple names: authors list (link) - Ainsworth BE, Haskell WL, Whitt MC, Irwin ML, Swartz AM, Strath SJ, O'Brien WL, Bassett DR, Schmitz KH, Emplaincourt PO, Jacobs DR, Leon AS; Haskell; Whitt; Irwin; Swartz; Strath; O'Brien; Bassett Jr; Schmitz; Emplaincourt; Jacobs Jr; Leon (2000). "Compendium of physical activities: an update of activity codes and MET intensities". Med Sci Sports Exerc. 32 (9 Suppl): S498–504. doi:10.1097/00005768-200009001-00009. PMID 10993420.
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: CS1 maint: multiple names: authors list (link) - Ainsworth BE, Haskell WL, Herrmann SD, Meckes N, Bassett DR, Tudor-Locke C, Greer JL, Vezina J, Whitt-Glover MC, Leon AS; Haskell; Herrmann; Meckes; Bassett Jr; Tudor-Locke; Greer; Vezina; Whitt-Glover; Leon (2011). "2011 Compendium of Physical Activities: a second update of codes and MET values". Med Sci Sports Exerc. 43 (8): 1575–81. doi:10.1249/MSS.0b013e31821ece12. PMID 21681120.
{{cite journal}}
: CS1 maint: multiple names: authors list (link) - Ainsworth BE, Haskell WL, Herrmann SD, Meckes N, Bassett Jr DR, Tudor-Locke C, Greer JL, Vezina J, Whitt-Glover MC, Leon AS. The Compendium of Physical Activities Tracking Guide. Healthy Lifestyles Research Center, College of Nursing & Health Innovation, Arizona State University. Retrieved [date] from the World Wide Web. https://sites.google.com/site/compendiumofphysicalactivities/
External links
- Media related to Physical exercises at Wikimedia Commons
- MedLinePlus's Topic on Exercise and Physical Fitness
- Science Daily's reference on Physical Exercise
- Guidance on the promotion and creation of physical environments that support increased levels of physical activity."Physical activity and the environment".