Jump to content

Eunjoon Kim

From Wikipedia, the free encyclopedia

This is an old revision of this page, as edited by Rickinasia (talk | contribs) at 07:37, 18 April 2018 (Category). The present address (URL) is a permanent link to this revision, which may differ significantly from the current revision.

Eunjoon Kim
김은준 교수
BornJan 20, 1964
NationalityKorea
Alma materMichigan State University
Scientific career
FieldsDepartment of Neuroscience
InstitutionsKAIST
Doctoral advisorJames L. Bennett

Eunjoon Kim is a professor of KAIST and director of Center for Synaptic Brain Dysfunctions within the Institute for Basic Science (IBS). He graduated from Busan National University in 1986, received master's degree at KAIST in 1988, received PhD degree at Michigan State University in 1994, and worked at Harvard Medical School as a postdoctoral fellow during 1995-1996. His current researches focus on molecular organization of neuronal synapses and synapse dysfunction-related psychiatric disorders.

Awards

  • 2013. POSCO Chungam Award, POSCO
  • 2012. Inchon Award, Dong-A Ilbo
  • 2012. Life Science Award, Korean Society for Molecular and Cell Biology
  • 2011. Best Research Award, KAIST[1]

Education

Work

  • 1988. 3 – 1991. 8 Research Associate, Korea Research Institute of Bioscience and Biotechnology, Daejeon, Korea
  • 1995. 1 – 1997. 2 Postdoc, Dept of Neurobiology and Howard Hughes Medical Institute, Harvard Medical School.P.I.: Morgan Sheng
  • 1997. 3 – 2000. 2 Assistant Professor, Dept of Pharmacology, Busan National University, Korea.
  • 2000. 3 – 2014 Assistant, Associate, & Full Professor, Dept of Biol. Sci., KAIST, Korea.
  • 2003. 7 – 2012.5 Director, National Creative Research Initiative Center for Synaptogenesis, Korea
  • 2012. 6 – present Director, Center for Synaptic Brain Dysfunctions, Institute for Basic Science (IBS), KAIST, Korea[3]

References

  1. ^ Eunjoon, Kim. "Google Scholar". Google Scholar.
  2. ^ Kim, Eunjoon. "Michigan State University".
  3. ^ Eunjoon, Kim (2012). "Autistic-like social behavior in Shank2-mutant mice improved by restoring NMDA receptor function". Nature. 486: 261–265. doi:10.1038/nature11208. PMID 22699620.