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Eunjoon Kim

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Eunjoon Kim
BornJan 20, 1964
NationalityKorea
Alma materBusan National University, KAIST, Michigan State University
Scientific career
FieldsNeuroscience, autism, protein receptors
InstitutionsKAIST, Institute for Basic Science
Doctoral advisorJames L. Bennett
Korean name
Hangul
Revised RomanizationGim Eunjun
McCune–ReischauerKim Ŭnchun
WebsiteIBS Center for Synaptic Brain Dysfunctions

Eunjoon Kim is a professor of KAIST and director of Center for Synaptic Brain Dysfunctions within the Institute for Basic Science (IBS). With over 150 publications to his name, his research has been cited over 16,000 times giving him an h-index of 61 and i10-index of 121.[1] He graduated from Busan National University in 1986, received master's degree at KAIST in 1988, received PhD degree at Michigan State University in 1994, and worked at Harvard Medical School as a postdoctoral fellow during 1995-1996. His current research focuses on molecular organization of neuronal synapses and synapse dysfunction-related psychiatric disorders.

Education

Work

  • 1988. 3 – 1991. 8 Research Associate, Korea Research Institute of Bioscience and Biotechnology, Daejeon, Korea
  • 1995. 1 – 1997. 2 Postdoc, Dept of Neurobiology and Howard Hughes Medical Institute, Harvard Medical School.P.I.: Morgan Sheng
  • 1997. 3 – 2000. 2 Assistant Professor, Dept of Pharmacology, Busan National University, Korea.
  • 2000. 3 – 2014 Assistant, Associate, & Full Professor, Dept of Biol. Sci., KAIST, Korea.
  • 2003. 7 – 2012.5 Director, National Creative Research Initiative Center for Synaptogenesis, Korea
  • 2012. 6 – present Director, Center for Synaptic Brain Dysfunctions, Institute for Basic Science, KAIST, Korea[3]

Awards

  • 2013: POSCO Chungam Award, POSCO
  • 2012: Inchon Award, Dong-A Ilbo
  • 2012: Life Science Award, Korean Society for Molecular and Cell Biology
  • 2011: Best Research Award, KAIST

References

  1. ^ Eunjoon, Kim. "Google Scholar". Google Scholar.
  2. ^ Kim, Eunjoon. "Michigan State University".
  3. ^ Eunjoon, Kim (2012). "Autistic-like social behavior in Shank2-mutant mice improved by restoring NMDA receptor function". Nature. 486: 261–265. doi:10.1038/nature11208. PMID 22699620.