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This is an old revision of this page, as edited by Joannamasel (talk | contribs) at 21:25, 3 September 2018 (Aging can't be beaten?). The present address (URL) is a permanent link to this revision, which may differ significantly from the current revision.

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The origin of senescence

The great majority of species in the phylum, chordata, are subject to death from aging-associated disease. Single cell organisms are not. The section on "Theories of aging" suggests that some instances of senescence occurring within a species evolved as a preventative for cancer, but gives no reference. Is this as much as can be had in Wikipedia on the origin of senescence? - Fartherred (talk) 18:31, 4 March 2016 (UTC)[reply]

Too technical/too much jargon

Much of this article is greatly informative but I also found myself skipping large swaths and entire sections that were too technical and filled with abbreviated jargon. Geeks On Hugs (talk) 01:55, 28 December 2016 (UTC)[reply]

Aging can't be beaten?

There's a study from Paul Nelson, professor of ecology and evolutionary biology and at the University of Arizona, and Joanna Masel, a postdoc researcher from the same university, stating that mathematically, it is impossible to beat aging, and their study is titled as "Intercellular competition and the inevitability of multicellular aging"

http://www.pnas.org/content/114/49/12982

Should we add this to the article?--EPN-001GF IZEN བཀྲ་ཤིས་བདེ་ལེགས། 08:52, 27 January 2018 (UTC)[reply]

Done. Although I am the professor and he is the postdoc :-) Joannamasel (talk) 21:24, 3 September 2018 (UTC)[reply]

Hatnote

Currently the hatnote reads, This article is about the aging of whole organisms including animals. For the state of cellular growth arrest and aberrant secretory phenotype, see cellular senescence. For aging specifically in humans, see aging. For the study of aging in humans, see gerontology. For the science of the care of the elderly, see geriatrics. For experimental gerontology, see life extension. For the stress- and age-related developmental aging phenomena in plants, see plant senescence. For premature aging disorders, see Progeroid syndromes. This seems excessive and contrary to guidelines. MOS:HATNOTE says hatnotes are "short notes" and "limit hatnotes to just one at the top of the page". This one is eight sentences disambiguating many different topics. Any suggestions for simplifying this? Are the linked topics suitable for a single disambiguation page? Deli nk (talk) 19:05, 15 May 2018 (UTC)[reply]

@Deli nk: I agree: the hatnote is unnecessarily long and unduly separates the reader from content. Most of the hatnote references are really more suitable for the See also section, so I have added them there (if they weren't there already). Cellular senescence is mentioned early in the lead, is in the title of 2 section headings, and has a hatnote already at a section. I have retained 2 redirects so the hatnote now reads "This article is about the aging of whole organisms including animals. For aging specifically in humans, see Aging. For aging specifically in plants, see Plant senescence.". Shhhnotsoloud (talk) 13:56, 17 August 2018 (UTC)[reply]