File:Gitelman syndrome.jpg
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| Description | A model of transport mechanisms in the distal convoluted tubule. Sodium-chloride (NaCl) enters the cell via the apical thiazide-sensitive NCC and leaves the cell through the basolateral Cl- channel (ClC-Kb), and the Na+/K+-ATPase. Indicated also are the recently identified magnesium channel TRPM6 in the apical membrane, and a putative Na/Mg exchanger in the basolateral membrane. These transport mechanisms play a role in familial hypokalemia-hypomagnesemia or Gitelman's syndrome. |
| Date | Published: 30 July 2008 |
| Source | Orphanet Journal of Rare Diseases 2008, 3:22. doi:10.1186/1750-1172-3-22 |
| Author | Nine VAM Knoers and Elena N Levtchenko |
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| Date/Time | Thumbnail | Dimensions | User | Comment | |
|---|---|---|---|---|---|
| current | 19:52, 21 December 2008 | | 656 × 650 (74 KB) | Stevenfruitsmaak | {{Information |Description=A model of transport mechanisms in the distal convoluted tubule. Sodium-chloride (NaCl) enters the cell via the apical thiazide-sensitive NCC and leaves the cell through the basolateral Cl- channel (ClC-Kb), and the Na+/K+-ATPas |
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